Gene Information

Gene symbol: ACTB

Gene name: actin, beta

HGNC ID: 132

Related Genes

# Gene Symbol Number of hits
1 AAVS1 1 hits
2 BRCA1 1 hits
3 BRCA2 1 hits
4 CD44 1 hits
5 CDKN1C 1 hits
6 CSF1 1 hits
7 DDIT3 1 hits
8 DYNC1H1 1 hits
9 EGFR 1 hits
10 FOS 1 hits
11 GPR180 1 hits
12 HIST1H2BO 1 hits
13 HIST2H2BE 1 hits
14 HRAS 1 hits
15 HSPG2 1 hits
16 IGF1 1 hits
17 IGFALS 1 hits
18 IGFBP2 1 hits
19 IL2 1 hits
20 INS 1 hits
21 MDK 1 hits
22 MLLT3 1 hits
23 MYC 1 hits
24 SLC9A3R2 1 hits
25 SMARCA1 1 hits
26 TP63 1 hits

Related Sentences

# PMID Sentence
1 9032395 The Rep proteins of adeno-associated virus type 2 (AAV) are known to bind to Rep recognition sequences (RRSs) in the AAV inverted terminal repeats (ITRs), the AAV p5 promoter, and the preferred AAV integration site in human chromosome 19, called AAVS1.
2 9032395 We used the 16-mer core sequences of the RRSs in the AAV ITRs and AAVS1 separately as query sequences and identified 18 new RRSs in or flanking the genes coding for the following: tyrosine kinase activator protein 1 (TKA-1); colony stimulating factor-1; insulin-like growth factor binding protein 2 (IGFBP-2); histone H2B.1; basement membrane heparan sulfate proteoglycan, also known as perlecan; the AF-9 gene product, which is involved in the chromosomal translocation t (9:11)(p22:q23); the betaB subunit of the hormone known as inhibin; interleukin-2 enhancer binding factor; an endoplasmic reticulum-Golgi intermediate compartment resident protein called p63; a global transcription activator (hSNF2L); the beta-actin repair domain; a retinoic acid-inducible factor, also known as midkine; a breast tumor autoantigen; a growth-arrest- and DNA-damage-inducible protein called gadd45; the cyclin-dependent kinase inhibitor called KIP2, which inhibits several G1 cyclin-cyclin-dependent kinase complexes; and the hereditary breast and ovarian cancer gene (BRCA1).
3 9504854 Fourteen cases of DCIS and 11 of DCIS with minimal invasion were analysed for mRNA levels of beta-actin, EGFR, c-cerbB2, MTS1, k-ras, RB, BRCA1, cyclin E, and c-myc genes.
4 10375022 To understand the mechanism underlying the inhibition of proliferation by estradiol, we examined the expression of several growth related endogenous genes. c-Myc protooncogene expression was strongly inhibited by treatment with estradiol as was expression of BRCA1 and BRCA2 genes, which is in agreement with their mitogenic-dependent expression, while expression of beta-actin, a housekeeping gene, was not affected by hormone treatment.