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Gene Information

Gene symbol: CDK4

Gene name: cyclin-dependent kinase 4

HGNC ID: 1773

Synonyms: PSK-J3

Related Genes

# Gene Symbol Number of hits
1 DDIT3 1 hits
2 GLI1 1 hits
3 IFNG 1 hits
4 LRP1 1 hits
5 MDM2 1 hits
6 RAP1B 1 hits
7 TSPAN31 1 hits

Related Sentences

# PMID Sentence
1 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
2 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
3 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
4 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
5 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
6 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
7 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
8 8912848 Refined mapping of 12q13-q15 amplicons in human malignant gliomas suggests CDK4/SAS and MDM2 as independent amplification targets.
9 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
10 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
11 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
12 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
13 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
14 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
15 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
16 8912848 The genes most frequently amplified and overexpressed were CDK4 (with coamplification of SAS) and MDM2.
17 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
18 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
19 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
20 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
21 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
22 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
23 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
24 8912848 Because individual malignant gliomas showed CDK4/SAS amplification but no MDM2 amplification and vice versa, the possibility remained of a common amplification target gene located between CDK4 and MDM2.
25 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
26 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
27 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
28 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
29 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
30 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
31 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
32 8912848 All tumors and cell lines were analyzed at eight gene loci and six anonymous loci from 12q13-q15, including seven loci located between CDK4 and MDM2.
33 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
34 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
35 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
36 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
37 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
38 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
39 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
40 8912848 These studies revealed two centers of amplification, one at CDK4/SAS and the other at MDM2.
41 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
42 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
43 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
44 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
45 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
46 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
47 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
48 8912848 A number of loci located close to either MDM2 or CDK4/SAS, including the genes GADD153, GLI, RAP1B, A2MR, and IFNG, were found to be coamplified in some tumors but not overexpressed consistently.
49 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
50 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
51 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
52 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
53 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
54 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
55 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
56 8912848 All amplicons were discontinuous between CDK4/SAS and MDM2.
57 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
58 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
59 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
60 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
61 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
62 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
63 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
64 8912848 Our results thus exclude a common amplification target between CDK4/SAS and MDM2 and provide additional evidence that these genes represent two independent targets of selection.
65 9772286 We explored the relative densitometric measure of a 119 bp fragment of the CDK4 gene versus an 82 bp fragment of the IFNG gene.
66 10203608 We explored the relative densitometric measure of a 119 bp fragment of the CDK4 gene versus an 82 bp fragment of the IFNG gene.