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Gene Information
Gene symbol: GPR146
Gene name:
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | HES1 | 1 hits |
| 2 | IFNG | 1 hits |
| 3 | IRF3 | 1 hits |
| 4 | STAT1 | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 28464285 | Elimination of GPR146-mediated antiviral function through IRF3/HES1-signalling pathway. |
| 2 | 28464285 | Here, we demonstrated that the expression of G-protein-coupled receptor 146 (GPR146) is highly increased by both IFN-β and IFN-γ in a signal transducer and activator of transcription 1-dependent signalling pathway. |
| 3 | 28464285 | Surprisingly, virus-activated IFN regulatory factor 3 (IRF3) signalling not only induces the expression of IFN but also represses GPR146 expression through HES1 (hairy and enhancer of split-1)-mediated transcriptional activity to establish a dynamic equilibrium between pro-viral and antiviral stages in host cells. |
| 4 | 28464285 | Taken together, these data reveal the antiviral role of GPR146 in fighting viral infection although the GPR146-mediated protection is eliminated by IRF3/HES1-signalling, which suggests a potential therapeutic significance of both GPR146 and HES1 signalling in viral infection. |
| 5 | 28464285 | Elimination of GPR146-mediated antiviral function through IRF3/HES1-signalling pathway. |
| 6 | 28464285 | Here, we demonstrated that the expression of G-protein-coupled receptor 146 (GPR146) is highly increased by both IFN-β and IFN-γ in a signal transducer and activator of transcription 1-dependent signalling pathway. |
| 7 | 28464285 | Surprisingly, virus-activated IFN regulatory factor 3 (IRF3) signalling not only induces the expression of IFN but also represses GPR146 expression through HES1 (hairy and enhancer of split-1)-mediated transcriptional activity to establish a dynamic equilibrium between pro-viral and antiviral stages in host cells. |
| 8 | 28464285 | Taken together, these data reveal the antiviral role of GPR146 in fighting viral infection although the GPR146-mediated protection is eliminated by IRF3/HES1-signalling, which suggests a potential therapeutic significance of both GPR146 and HES1 signalling in viral infection. |
| 9 | 28464285 | Elimination of GPR146-mediated antiviral function through IRF3/HES1-signalling pathway. |
| 10 | 28464285 | Here, we demonstrated that the expression of G-protein-coupled receptor 146 (GPR146) is highly increased by both IFN-β and IFN-γ in a signal transducer and activator of transcription 1-dependent signalling pathway. |
| 11 | 28464285 | Surprisingly, virus-activated IFN regulatory factor 3 (IRF3) signalling not only induces the expression of IFN but also represses GPR146 expression through HES1 (hairy and enhancer of split-1)-mediated transcriptional activity to establish a dynamic equilibrium between pro-viral and antiviral stages in host cells. |
| 12 | 28464285 | Taken together, these data reveal the antiviral role of GPR146 in fighting viral infection although the GPR146-mediated protection is eliminated by IRF3/HES1-signalling, which suggests a potential therapeutic significance of both GPR146 and HES1 signalling in viral infection. |
| 13 | 28464285 | Elimination of GPR146-mediated antiviral function through IRF3/HES1-signalling pathway. |
| 14 | 28464285 | Here, we demonstrated that the expression of G-protein-coupled receptor 146 (GPR146) is highly increased by both IFN-β and IFN-γ in a signal transducer and activator of transcription 1-dependent signalling pathway. |
| 15 | 28464285 | Surprisingly, virus-activated IFN regulatory factor 3 (IRF3) signalling not only induces the expression of IFN but also represses GPR146 expression through HES1 (hairy and enhancer of split-1)-mediated transcriptional activity to establish a dynamic equilibrium between pro-viral and antiviral stages in host cells. |
| 16 | 28464285 | Taken together, these data reveal the antiviral role of GPR146 in fighting viral infection although the GPR146-mediated protection is eliminated by IRF3/HES1-signalling, which suggests a potential therapeutic significance of both GPR146 and HES1 signalling in viral infection. |