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Gene Information
Gene symbol: IL29
Gene name: interleukin 29 (interferon, lambda 1)
HGNC ID: 18363
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | CCL5 | 1 hits |
| 2 | CXCL10 | 1 hits |
| 3 | IFNA1 | 1 hits |
| 4 | IFNA2 | 1 hits |
| 5 | IFNB1 | 1 hits |
| 6 | IFNG | 1 hits |
| 7 | IL28A | 1 hits |
| 8 | IL28B | 1 hits |
| 9 | STAT1 | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 26020282 | Type I IFNs (IFNA1, IFNB1), type II (IFNG), type III (IFNL1, IFNL2/3), IFNL4 and ISG hepatic expressions were measured by qPCR from in 65 chronic hepatitis C (CHC) patients whose IFNL4-associated rs368234815 and IFNL3-associated rs12989760 genotype were determined. |
| 2 | 26020282 | Levels of ISGs and IFNL2/3 mRNAs were lower in IFNL3 rs12979860 CC patients compared with non-CC patients, and in treatment responders, compared with nonresponders. |
| 3 | 26020282 | Hepatic levels of ISGs in CHC are associated with IFNL2/3 and IFNL4 expression, suggesting that IFNLs, not other types of IFNs, drive ISG expression. |
| 4 | 26020282 | Hepatic IFNL2/3 expression is functionally linked to IFNL4 and IFNL3 polymorphisms, potentially explaining the tight association among ISG expression and treatment response. |
| 5 | 26840694 | Hepatic gene expression of type III IFNs (IFNL1, IFNL3, IFNL4-ΔG) similarly decreased with treatment, while IFNA2 expression, undetectable in all subjects pretreatment, was detected post-treatment in three subjects who achieved a SVR. |
| 6 | 26840694 | Other IFNs had no change in gene expression (IFNG, IFNB1, IFNA5) or could not be detected. |
| 7 | 26869670 | Viral titres were measured by 50% tissue culture infective dose and release of interferon-γ-induced protein 10 (IP-10) and RANTES protein assessed by ELISA. |
| 8 | 26869670 | Using cultures from healthy donors, enhanced STAT-1 phosphorylation upon inhibition of Pim1 kinase activity resulted in increased mRNA expression of interferon-β, interleukin-29, IP-10 and RANTES 12 h after infection and increased protein levels of IP-10 and RANTES 24 h after infection.We have identified Pim1 kinase as novel target to reduce viral replication in ALI cultures of PBECs. |