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Gene Information
Gene symbol: NGF
Gene name: nerve growth factor (beta polypeptide)
HGNC ID: 7808
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | CHAT | 1 hits |
| 2 | IFNG | 1 hits |
| 3 | IGF1 | 1 hits |
| 4 | INS | 1 hits |
| 5 | TGFA | 1 hits |
| 6 | TGFB1 | 1 hits |
| 7 | TNF | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 19129516 | The present study was conducted to explore the source of acetylcholine (ACH) in the corpus luteum (CL) and to test our hypothesis of an antiapoptotic role of ACH in the bovine CL and, further, to investigate whether nerve growth factor (NGF), insulin-like growth factor 1 (IGF1), and transforming growth factor beta1 (TGFB1) influence the expression of choline acetyltransferase (CHAT), the biosynthetic enzyme of ACH, in cultured bovine luteal cells. |
| 2 | 19129516 | The present study was conducted to explore the source of acetylcholine (ACH) in the corpus luteum (CL) and to test our hypothesis of an antiapoptotic role of ACH in the bovine CL and, further, to investigate whether nerve growth factor (NGF), insulin-like growth factor 1 (IGF1), and transforming growth factor beta1 (TGFB1) influence the expression of choline acetyltransferase (CHAT), the biosynthetic enzyme of ACH, in cultured bovine luteal cells. |
| 3 | 19129516 | The present study was conducted to explore the source of acetylcholine (ACH) in the corpus luteum (CL) and to test our hypothesis of an antiapoptotic role of ACH in the bovine CL and, further, to investigate whether nerve growth factor (NGF), insulin-like growth factor 1 (IGF1), and transforming growth factor beta1 (TGFB1) influence the expression of choline acetyltransferase (CHAT), the biosynthetic enzyme of ACH, in cultured bovine luteal cells. |
| 4 | 19129516 | Cell viability and TUNEL assays were performed on cultured midluteal cells treated with or without tumor necrosis factor alpha (TNF)/interferon gamma (IFNG) in the presence of ACH and its muscarinic (atropine) and nicotinic (mecamylamine) receptor antagonists. |
| 5 | 19129516 | Cell viability and TUNEL assays were performed on cultured midluteal cells treated with or without tumor necrosis factor alpha (TNF)/interferon gamma (IFNG) in the presence of ACH and its muscarinic (atropine) and nicotinic (mecamylamine) receptor antagonists. |
| 6 | 19129516 | Cell viability and TUNEL assays were performed on cultured midluteal cells treated with or without tumor necrosis factor alpha (TNF)/interferon gamma (IFNG) in the presence of ACH and its muscarinic (atropine) and nicotinic (mecamylamine) receptor antagonists. |
| 7 | 19129516 | ACH increased cell viability and prevented cell death induced by TNF/IFNG. |
| 8 | 19129516 | ACH increased cell viability and prevented cell death induced by TNF/IFNG. |
| 9 | 19129516 | ACH increased cell viability and prevented cell death induced by TNF/IFNG. |
| 10 | 19129516 | TNF/IFNG treatment downregulated CHAT expression, whereas NGF, IGF1, and TGFB1 upregulated CHAT expression, in cultured luteal cells. |
| 11 | 19129516 | TNF/IFNG treatment downregulated CHAT expression, whereas NGF, IGF1, and TGFB1 upregulated CHAT expression, in cultured luteal cells. |
| 12 | 19129516 | TNF/IFNG treatment downregulated CHAT expression, whereas NGF, IGF1, and TGFB1 upregulated CHAT expression, in cultured luteal cells. |
| 13 | 19129516 | Locally produced ACH appears to be regulated by NGF, IGF1, and TGFB1. |
| 14 | 19129516 | Locally produced ACH appears to be regulated by NGF, IGF1, and TGFB1. |
| 15 | 19129516 | Locally produced ACH appears to be regulated by NGF, IGF1, and TGFB1. |