# |
PMID |
Sentence |
1 |
23941776
|
Role of tumor necrosis factor-α, interferon-γ and Fas-ligand on in vitro nitric oxide activity in the corpus luteum.
|
2 |
23941776
|
Role of tumor necrosis factor-α, interferon-γ and Fas-ligand on in vitro nitric oxide activity in the corpus luteum.
|
3 |
23941776
|
Role of tumor necrosis factor-α, interferon-γ and Fas-ligand on in vitro nitric oxide activity in the corpus luteum.
|
4 |
23941776
|
Moreover, TNF, IFNG and FASL were shown to regulate equine CL establishment and regression.
|
5 |
23941776
|
Moreover, TNF, IFNG and FASL were shown to regulate equine CL establishment and regression.
|
6 |
23941776
|
Moreover, TNF, IFNG and FASL were shown to regulate equine CL establishment and regression.
|
7 |
23941776
|
TNF increased eNOS mRNA level and NO metabolite (nitrite) production during CL growth.
|
8 |
23941776
|
TNF increased eNOS mRNA level and NO metabolite (nitrite) production during CL growth.
|
9 |
23941776
|
TNF increased eNOS mRNA level and NO metabolite (nitrite) production during CL growth.
|
10 |
23941776
|
Cytokines combined action (TNF+IFNG+FASL) promoted eNOS protein upregulation in mid-CL and nitrite production in mid and late-CL.
|
11 |
23941776
|
Cytokines combined action (TNF+IFNG+FASL) promoted eNOS protein upregulation in mid-CL and nitrite production in mid and late-CL.
|
12 |
23941776
|
Cytokines combined action (TNF+IFNG+FASL) promoted eNOS protein upregulation in mid-CL and nitrite production in mid and late-CL.
|
13 |
23941776
|
These results indicate that in equine CL, cytokines TNF, IFNG and FASL regulate NO activity, via eNOS expression modulation.
|
14 |
23941776
|
These results indicate that in equine CL, cytokines TNF, IFNG and FASL regulate NO activity, via eNOS expression modulation.
|
15 |
23941776
|
These results indicate that in equine CL, cytokines TNF, IFNG and FASL regulate NO activity, via eNOS expression modulation.
|