Ignet

Gene Information

Gene symbol: BID

Gene name: BH3 interacting domain death agonist

HGNC ID: 1050

Related Genes

#	Gene Symbol	Number of hits
1	CASP8	1 hits
2	CASP9	1 hits
3	CTSB	1 hits

Related Sentences

#	PMID	Sentence
1	17982689	Lysosomal hydrolase cathepsin B (CB) is involved in the apoptotic process and has a key role in breast cancer cell programmed death through the activation of a pro-apoptotic protein BID.
2	17982689	Truncated BID participates in the mitochondrial apoptotic pathway that involves the activation of pro-caspase 9.
3	17982689	Expression of CB, BID and pro-caspase 9 was determined by Western blotting.
4	17982689	The apoptosis of T24 and MB49 cell lines was mediated by activation of pro-caspase 9 and BID, both proteins are involved in mitochondrial apoptosis.
5	17982689	Apoptosis and activation of pro-caspase 9 and BID were inhibited by CA-074Me (CA), a cell permeable CB inhibitor.
6	17982689	Lysosomal hydrolase cathepsin B (CB) is involved in the apoptotic process and has a key role in breast cancer cell programmed death through the activation of a pro-apoptotic protein BID.
7	17982689	Truncated BID participates in the mitochondrial apoptotic pathway that involves the activation of pro-caspase 9.
8	17982689	Expression of CB, BID and pro-caspase 9 was determined by Western blotting.
9	17982689	The apoptosis of T24 and MB49 cell lines was mediated by activation of pro-caspase 9 and BID, both proteins are involved in mitochondrial apoptosis.
10	17982689	Apoptosis and activation of pro-caspase 9 and BID were inhibited by CA-074Me (CA), a cell permeable CB inhibitor.
11	17982689	Lysosomal hydrolase cathepsin B (CB) is involved in the apoptotic process and has a key role in breast cancer cell programmed death through the activation of a pro-apoptotic protein BID.
12	17982689	Truncated BID participates in the mitochondrial apoptotic pathway that involves the activation of pro-caspase 9.
13	17982689	Expression of CB, BID and pro-caspase 9 was determined by Western blotting.
14	17982689	The apoptosis of T24 and MB49 cell lines was mediated by activation of pro-caspase 9 and BID, both proteins are involved in mitochondrial apoptosis.
15	17982689	Apoptosis and activation of pro-caspase 9 and BID were inhibited by CA-074Me (CA), a cell permeable CB inhibitor.
16	17982689	Lysosomal hydrolase cathepsin B (CB) is involved in the apoptotic process and has a key role in breast cancer cell programmed death through the activation of a pro-apoptotic protein BID.
17	17982689	Truncated BID participates in the mitochondrial apoptotic pathway that involves the activation of pro-caspase 9.
18	17982689	Expression of CB, BID and pro-caspase 9 was determined by Western blotting.
19	17982689	The apoptosis of T24 and MB49 cell lines was mediated by activation of pro-caspase 9 and BID, both proteins are involved in mitochondrial apoptosis.
20	17982689	Apoptosis and activation of pro-caspase 9 and BID were inhibited by CA-074Me (CA), a cell permeable CB inhibitor.
21	17982689	Lysosomal hydrolase cathepsin B (CB) is involved in the apoptotic process and has a key role in breast cancer cell programmed death through the activation of a pro-apoptotic protein BID.
22	17982689	Truncated BID participates in the mitochondrial apoptotic pathway that involves the activation of pro-caspase 9.
23	17982689	Expression of CB, BID and pro-caspase 9 was determined by Western blotting.
24	17982689	The apoptosis of T24 and MB49 cell lines was mediated by activation of pro-caspase 9 and BID, both proteins are involved in mitochondrial apoptosis.
25	17982689	Apoptosis and activation of pro-caspase 9 and BID were inhibited by CA-074Me (CA), a cell permeable CB inhibitor.
26	25443632	SHARPIN regulates immune signaling and contributes to full transcriptional activity and prevention of cell death in response to TNF in vitro.
27	25443632	The inactivating mouse Sharpin cpdm mutation causes TNF-dependent multi-organ inflammation, characterized by dermatitis, liver inflammation, splenomegaly, and loss of Peyer's patches.
28	25443632	TNFR1-induced apoptosis can proceed through caspase-8 and BID, but reduction in or loss of these players generally did not suppress inflammation, although Casp8 heterozygosity significantly delayed dermatitis.
29	25443632	Ripk3 or Mlkl deficiency partially ameliorated the multi-organ phenotype, and combined Ripk3 deletion and Casp8 heterozygosity almost completely suppressed it, even restoring Peyer's patches.
30	25443632	Unexpectedly, Sharpin, Ripk3 and Casp8 triple deficiency caused perinatal lethality.