Ignet

Gene Information

Gene symbol: GOLGA6

Gene name: golgi autoantigen, golgin subfamily a, 6

HGNC ID: 13567

Related Genes

#	Gene Symbol	Number of hits
1	EHMT2	1 hits
2	NT5C2	1 hits

Related Sentences

#	PMID	Sentence
1	11178400	[Abnormal toxicity - a relevant safety test under GLP- and GMP-conditions in the production of vaccines?]
2	11178400	This paper deals with the relevance of abnormal toxicity after the introduction of GMP- and GLP-conditions in the production of veterinary biologicals.
3	11178400	[Abnormal toxicity - a relevant safety test under GLP- and GMP-conditions in the production of vaccines?]
4	11178400	This paper deals with the relevance of abnormal toxicity after the introduction of GMP- and GLP-conditions in the production of veterinary biologicals.
5	11178440	The purpose of this project was to investigate the relevance of ATT after the introduction of GMP- and GLP-principles in the manufacturing of biological products.
6	15496321	Thus it has become imperative that, in concert with other quality control measures, a potency test be utilized for the GMP/GLP lot-release of DC products for preclinical and clinical studies.
7	23629948	Affinity purification also was used to identify the distinct composition of the h-CDYLa or h-CDYLb protein complex. h-CDYLb was found in a multiprotein complex with G9a and GLP, while the h-CDYLa complex did not contain these two enzymes.
8	25483693	To meet the time constraint imposed on this project, we used a distributed R&D model involving experts in the fields of protein engineering and production, bioinformatics, peptide synthesis/design and GMP/GLP manufacturing and testing standards.
9	25637356	GLP and G9a are major H3K9 dimethylases and are essential for mouse early embryonic development.
10	25637356	GLP and G9a both harbor ankyrin repeat domains that are capable of binding H3K9 methylation.
11	25637356	Here, we report that the histone methyltransferase activities of GLP and G9a are stimulated by neighboring nucleosomes that are premethylated at H3K9.
12	25637356	These stimulation events function in cis and are dependent on the H3K9 methylation binding activities of ankyrin repeat domains of GLP and G9a.
13	25637356	In mouse embryonic stem cells (ESCs) harboring a mutant GLP that lacks H3K9me1-binding activity, critical pluripotent genes, including Oct4 and Nanog, display inefficient establishment of H3K9me2 and delayed gene silencing during differentiation.
14	25637356	Collectively, our study reveals a new activation mechanism for GLP and G9a that plays an important role in ESC differentiation and mouse viability.
15	25637356	GLP and G9a are major H3K9 dimethylases and are essential for mouse early embryonic development.
16	25637356	GLP and G9a both harbor ankyrin repeat domains that are capable of binding H3K9 methylation.
17	25637356	Here, we report that the histone methyltransferase activities of GLP and G9a are stimulated by neighboring nucleosomes that are premethylated at H3K9.
18	25637356	These stimulation events function in cis and are dependent on the H3K9 methylation binding activities of ankyrin repeat domains of GLP and G9a.
19	25637356	In mouse embryonic stem cells (ESCs) harboring a mutant GLP that lacks H3K9me1-binding activity, critical pluripotent genes, including Oct4 and Nanog, display inefficient establishment of H3K9me2 and delayed gene silencing during differentiation.
20	25637356	Collectively, our study reveals a new activation mechanism for GLP and G9a that plays an important role in ESC differentiation and mouse viability.
21	25637356	GLP and G9a are major H3K9 dimethylases and are essential for mouse early embryonic development.
22	25637356	GLP and G9a both harbor ankyrin repeat domains that are capable of binding H3K9 methylation.
23	25637356	Here, we report that the histone methyltransferase activities of GLP and G9a are stimulated by neighboring nucleosomes that are premethylated at H3K9.
24	25637356	These stimulation events function in cis and are dependent on the H3K9 methylation binding activities of ankyrin repeat domains of GLP and G9a.
25	25637356	In mouse embryonic stem cells (ESCs) harboring a mutant GLP that lacks H3K9me1-binding activity, critical pluripotent genes, including Oct4 and Nanog, display inefficient establishment of H3K9me2 and delayed gene silencing during differentiation.
26	25637356	Collectively, our study reveals a new activation mechanism for GLP and G9a that plays an important role in ESC differentiation and mouse viability.
27	25637356	GLP and G9a are major H3K9 dimethylases and are essential for mouse early embryonic development.
28	25637356	GLP and G9a both harbor ankyrin repeat domains that are capable of binding H3K9 methylation.
29	25637356	Here, we report that the histone methyltransferase activities of GLP and G9a are stimulated by neighboring nucleosomes that are premethylated at H3K9.
30	25637356	These stimulation events function in cis and are dependent on the H3K9 methylation binding activities of ankyrin repeat domains of GLP and G9a.
31	25637356	In mouse embryonic stem cells (ESCs) harboring a mutant GLP that lacks H3K9me1-binding activity, critical pluripotent genes, including Oct4 and Nanog, display inefficient establishment of H3K9me2 and delayed gene silencing during differentiation.
32	25637356	Collectively, our study reveals a new activation mechanism for GLP and G9a that plays an important role in ESC differentiation and mouse viability.
33	25637356	GLP and G9a are major H3K9 dimethylases and are essential for mouse early embryonic development.
34	25637356	GLP and G9a both harbor ankyrin repeat domains that are capable of binding H3K9 methylation.
35	25637356	Here, we report that the histone methyltransferase activities of GLP and G9a are stimulated by neighboring nucleosomes that are premethylated at H3K9.
36	25637356	These stimulation events function in cis and are dependent on the H3K9 methylation binding activities of ankyrin repeat domains of GLP and G9a.
37	25637356	In mouse embryonic stem cells (ESCs) harboring a mutant GLP that lacks H3K9me1-binding activity, critical pluripotent genes, including Oct4 and Nanog, display inefficient establishment of H3K9me2 and delayed gene silencing during differentiation.
38	25637356	Collectively, our study reveals a new activation mechanism for GLP and G9a that plays an important role in ESC differentiation and mouse viability.