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PMID |
Sentence |
1 |
2647882
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Oxidative responses of rabbit alveolar macrophages: comparative priming activities of MIF/MAF, sera, and serum components.
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2 |
2647882
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It was further established that pretreatment of normal AM for 3 or 18 hr with MIF/MAF preparations (serum-free), fetal bovine serum (FBS), or bovine serum albumin (BSA) exhibited marked increases in their CL responses following challenge with PMA.
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3 |
2647882
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Oxidative responses of rabbit alveolar macrophages: comparative priming activities of MIF/MAF, sera, and serum components.
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4 |
2647882
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It was further established that pretreatment of normal AM for 3 or 18 hr with MIF/MAF preparations (serum-free), fetal bovine serum (FBS), or bovine serum albumin (BSA) exhibited marked increases in their CL responses following challenge with PMA.
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5 |
3609455
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Purification of mediators resulted in separation of MIF/MAF and MCF 2.
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6 |
3609455
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Lipid metabolisms of macrophages stimulated with MIF/MAF and MCF are different 4.
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7 |
3609455
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MIF/MAF suppresses MCF-reaction through monokines.
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8 |
3609455
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Purification of mediators resulted in separation of MIF/MAF and MCF 2.
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9 |
3609455
|
Lipid metabolisms of macrophages stimulated with MIF/MAF and MCF are different 4.
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10 |
3609455
|
MIF/MAF suppresses MCF-reaction through monokines.
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11 |
3609455
|
Purification of mediators resulted in separation of MIF/MAF and MCF 2.
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12 |
3609455
|
Lipid metabolisms of macrophages stimulated with MIF/MAF and MCF are different 4.
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13 |
3609455
|
MIF/MAF suppresses MCF-reaction through monokines.
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14 |
22427637
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Bcl6 and Maf cooperate to instruct human follicular helper CD4 T cell differentiation.
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15 |
22427637
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The introduction of Bcl6 expression in primary human CD4 T cells resulted in the regulation of a core set of migration genes that enable trafficking to germinal centers: CXCR4, CXCR5, CCR7, and EBI2.
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16 |
22427637
|
Bcl6 expression also induced a module of protein expression critical for T-B interactions, including SAP, CD40L, PD-1, ICOS, and CXCL13.
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17 |
22427637
|
This constitutes direct evidence for Bcl6 control of most of these functions and includes three genes known to be loci of severe human genetic immunodeficiencies (CD40L, SH2D1A, and ICOS).
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18 |
22427637
|
Introduction of Bcl6 did not alter the expression of IL-21 or IL-4, the primary cytokines of human Tfh cells.
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19 |
22427637
|
Coexpression of Bcl6 and Maf revealed that Bcl6 and Maf cooperate in the induction of CXCR4, PD-1, and ICOS.
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20 |
22427637
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Altogether, these findings reveal that Bcl6 and Maf collaborate to orchestrate a suite of genes that define core characteristics of human Tfh cell biology.
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21 |
22427637
|
Bcl6 and Maf cooperate to instruct human follicular helper CD4 T cell differentiation.
|
22 |
22427637
|
The introduction of Bcl6 expression in primary human CD4 T cells resulted in the regulation of a core set of migration genes that enable trafficking to germinal centers: CXCR4, CXCR5, CCR7, and EBI2.
|
23 |
22427637
|
Bcl6 expression also induced a module of protein expression critical for T-B interactions, including SAP, CD40L, PD-1, ICOS, and CXCL13.
|
24 |
22427637
|
This constitutes direct evidence for Bcl6 control of most of these functions and includes three genes known to be loci of severe human genetic immunodeficiencies (CD40L, SH2D1A, and ICOS).
|
25 |
22427637
|
Introduction of Bcl6 did not alter the expression of IL-21 or IL-4, the primary cytokines of human Tfh cells.
|
26 |
22427637
|
Coexpression of Bcl6 and Maf revealed that Bcl6 and Maf cooperate in the induction of CXCR4, PD-1, and ICOS.
|
27 |
22427637
|
Altogether, these findings reveal that Bcl6 and Maf collaborate to orchestrate a suite of genes that define core characteristics of human Tfh cell biology.
|
28 |
22427637
|
Bcl6 and Maf cooperate to instruct human follicular helper CD4 T cell differentiation.
|
29 |
22427637
|
The introduction of Bcl6 expression in primary human CD4 T cells resulted in the regulation of a core set of migration genes that enable trafficking to germinal centers: CXCR4, CXCR5, CCR7, and EBI2.
|
30 |
22427637
|
Bcl6 expression also induced a module of protein expression critical for T-B interactions, including SAP, CD40L, PD-1, ICOS, and CXCL13.
|
31 |
22427637
|
This constitutes direct evidence for Bcl6 control of most of these functions and includes three genes known to be loci of severe human genetic immunodeficiencies (CD40L, SH2D1A, and ICOS).
|
32 |
22427637
|
Introduction of Bcl6 did not alter the expression of IL-21 or IL-4, the primary cytokines of human Tfh cells.
|
33 |
22427637
|
Coexpression of Bcl6 and Maf revealed that Bcl6 and Maf cooperate in the induction of CXCR4, PD-1, and ICOS.
|
34 |
22427637
|
Altogether, these findings reveal that Bcl6 and Maf collaborate to orchestrate a suite of genes that define core characteristics of human Tfh cell biology.
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35 |
23112821
|
We conducted full-exon sequencing in samples obtained from Uganda (n = 48) and South Africa (n = 48), in four genes in the TLR pathway: TLR2, TLR4, TLR6, and TIRAP.
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36 |
23112821
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We identified one novel TIRAP SNP (with minor allele frequency [MAF] 3.2%) and a novel TLR6 SNP (MAF 8%) in the Ugandan population, and a TLR6 SNP that is unique to the South African population (MAF 14%).
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