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Gene Information
Gene symbol: PF4V1
Gene name: platelet factor 4 variant 1
HGNC ID: 8862
Synonyms: SCYB4V1, CXCL4V1, CXCL4L1
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | CCL2 | 1 hits |
| 2 | CXCL10 | 1 hits |
| 3 | CXCL11 | 1 hits |
| 4 | IFNG | 1 hits |
| 5 | IL8 | 1 hits |
| 6 | PF4 | 1 hits |
| 7 | PPBP | 1 hits |
| 8 | VWF | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 17291641 | Effect of allergen-specific immunotherapy on plasma level of platelet factor 4 (PF-4) and beta-thromboglobulin (beta-TG), platelet activation markers in patients with house dust mite allergy. |
| 2 | 17291641 | Plasma levels of PF-4 and beta-TG, platelet activation markers were measured using commercial ELISA kit. |
| 3 | 17291641 | PF-4 and beta-TG basal levels were not significantly different between the patients and the healthy subjects. |
| 4 | 17291641 | Thus, it seems that the degree of platelet activation measured by PF-4 and beta-TG does not change in PAR patients during hyposensitization with mite allergen extracts, after the maximum dose was reached. |
| 5 | 19552626 | The specificity for human substrates is not restricted to IL-8, since we also detected in vitro protease activity for another CXC chemokine GRO-alpha (growth-related oncogene alpha), but not for NAP-2 (neutrophil-activating protein 2), SDF (stromal-cell-derived factor)-1alpha, PF-4 (platelet factor 4), I-TAC (interferon-gamma-inducible T-cell alpha-chemoattractant), IP-10 (interferon-gamma-inducible protein 10) and MCP-1 (monocyte chemoattractant protein 1). |
| 6 | 22966172 | In thrombotic thrombocytopenic purpura, the clinical impact of ADAMTS13 deficiency caused by autoantibodies likely depends on the balance between residual antigen, that is, enzyme activity, and demand imposed by local genesis of ultralarge multimers of von Willebrand factor. |
| 7 | 22966172 | A corollary of these concepts is that disrupting platelet factor 4 and β(2)GPI conformation (or ultralarge multimer of von Willebrand factor oligomerization or function) might provide a disease-targeted approach to prevent thrombosis without systemic anticoagulation or immunosuppression. |