Ignet

Gene Information

Gene symbol: PIK3CG

Gene name: phosphatidylinositol-4,5-bisphosphate 3-kinase, catalytic subunit gamma

HGNC ID: 8978

Related Genes

#	Gene Symbol	Number of hits
1	AKT1	1 hits
2	CXCL10	1 hits
3	IRF3	1 hits
4	NGF	1 hits
5	NLRP3	1 hits
6	PI3	1 hits
7	RHOD	1 hits

Related Sentences

#	PMID	Sentence
1	12369850	Other cellular consequences of RAS activation including interactions with the RHO-family proteins, the PI3-kinase pathway, and other mitogen activated protein kinase cascades, will be discussed.
2	21497908	Regulation of influenza A virus induced CXCL-10 gene expression requires PI3K/Akt pathway and IRF3 transcription factor.
3	21497908	To understand the regulation of CXCL-10, we investigated the role of PI3K/AKT pathway in regulating virus induced CXCL-10 production.
4	21497908	Previously we have shown that wild type (WT) influenza A virus infection activates PI3K/AKT pathway, whereas PR8-SH3-mf-1 mutant virus is unable to activate this pathway.
5	21497908	Our data suggested that PI3K/AKT pathway contributes to influenza A virus induced CXCL-10 production.
6	21497908	This process is involved in binding of IRF3 to the ISRE binding site in CXCL-10 promoter region.
7	21497908	Regulation of influenza A virus induced CXCL-10 gene expression requires PI3K/Akt pathway and IRF3 transcription factor.
8	21497908	To understand the regulation of CXCL-10, we investigated the role of PI3K/AKT pathway in regulating virus induced CXCL-10 production.
9	21497908	Previously we have shown that wild type (WT) influenza A virus infection activates PI3K/AKT pathway, whereas PR8-SH3-mf-1 mutant virus is unable to activate this pathway.
10	21497908	Our data suggested that PI3K/AKT pathway contributes to influenza A virus induced CXCL-10 production.
11	21497908	This process is involved in binding of IRF3 to the ISRE binding site in CXCL-10 promoter region.
12	21497908	Regulation of influenza A virus induced CXCL-10 gene expression requires PI3K/Akt pathway and IRF3 transcription factor.
13	21497908	To understand the regulation of CXCL-10, we investigated the role of PI3K/AKT pathway in regulating virus induced CXCL-10 production.
14	21497908	Previously we have shown that wild type (WT) influenza A virus infection activates PI3K/AKT pathway, whereas PR8-SH3-mf-1 mutant virus is unable to activate this pathway.
15	21497908	Our data suggested that PI3K/AKT pathway contributes to influenza A virus induced CXCL-10 production.
16	21497908	This process is involved in binding of IRF3 to the ISRE binding site in CXCL-10 promoter region.
17	21497908	Regulation of influenza A virus induced CXCL-10 gene expression requires PI3K/Akt pathway and IRF3 transcription factor.
18	21497908	To understand the regulation of CXCL-10, we investigated the role of PI3K/AKT pathway in regulating virus induced CXCL-10 production.
19	21497908	Previously we have shown that wild type (WT) influenza A virus infection activates PI3K/AKT pathway, whereas PR8-SH3-mf-1 mutant virus is unable to activate this pathway.
20	21497908	Our data suggested that PI3K/AKT pathway contributes to influenza A virus induced CXCL-10 production.
21	21497908	This process is involved in binding of IRF3 to the ISRE binding site in CXCL-10 promoter region.
22	22491318	Viral genomes are maintained at an average copy number of 25 per neuron and can be induced to productively replicate by interfering with PI3-Kinase / Akt signaling or the simple withdrawal of nerve growth factor(1).
23	24463269	Here, to provide an insight into its mode of action, recent findings regarding innate immune responses induced by alum and their impact on adaptive immunity are described, with a particular emphasis on early recognition of alum, including NLRP3 and PI3 kinase activation, adjuvant-induced cell death and the release of endogenous danger signals.