Ignet

Gene Information

Gene symbol: PIM2

Gene name: pim-2 oncogene

HGNC ID: 8987

Related Genes

#	Gene Symbol	Number of hits
1	BCL2A1	1 hits
2	CASP2	1 hits
3	CASP8	1 hits
4	CAST	1 hits
5	CCL4	1 hits
6	CDK2	1 hits
7	CFLAR	1 hits
8	GADD45B	1 hits
9	IL1B	1 hits
10	NFKB1	1 hits
11	NFKB2	1 hits
12	PIM1	1 hits
13	PIM3	1 hits
14	RELA	1 hits
15	SOD2	1 hits
16	TNFAIP3	1 hits
17	TNFRSF10C	1 hits
18	TNFRSF10D	1 hits
19	TNFSF10	1 hits
20	VEGFA	1 hits
21	XIAP	1 hits

Related Sentences

#	PMID	Sentence
1	19049470	Here, we report the efficient synthesis of all PIMs including phosphatidylinositol (PI) and phosphatidylinositol mono- to hexa-mannoside (PIM1 to PIM6).
2	19049470	The synthetic PIMs were immobilized on microarray slides to elucidate differences in binding to the dendritic cell specific intercellular adhesion molecule-grabbing nonintegrin (DC-SIGN) receptor.
3	22120192	The addition of synthetic PIM(2) to the vaccine resulted in a significant reduction in lung bacterial counts and a cytokine profile indicating a Th 1 type immune response.
4	22120192	The addition of the other PIM(2) derivatives to the vaccine or the fusion protein alone did not result in reduced lung bacterial counts; moreover, the addition of PIM(2)ME appeared to negate the induction of an antigen-specific interferon-γ response and protection against tuberculosis.
5	22120192	The addition of synthetic PIM(2) to the vaccine resulted in a significant reduction in lung bacterial counts and a cytokine profile indicating a Th 1 type immune response.
6	22120192	The addition of the other PIM(2) derivatives to the vaccine or the fusion protein alone did not result in reduced lung bacterial counts; moreover, the addition of PIM(2)ME appeared to negate the induction of an antigen-specific interferon-γ response and protection against tuberculosis.
7	22986450	Specific targets in this category included genes asso-ciated with the intrinsic and extrinsic apoptotic pathways (CFLAR, TNFAIP3, TNFRSF10D, SOD2, BCL2A1, BIRC4, PIM2, TNFSF10, TNFRSF10C, CASP2 and CASP8) and genes that act via the NFĸB pathway and other mechanisms to prolong cell viability (NFKB1, NFKB2 and RELA, IL1B, CAST, CDK2,GADD45B, BCL3, BIRC3, CDK2, IL1A, PBEF1, IL6, CXCL1, CCL4 and VEGF).
8	22986450	Moreover, we demonstrate that the X-linked inhibitor of apoptosis protein remained abundant in polymorphonuclear leukocytes over 48 h of LVS infection, whereas BAX mRNA and protein were progressively downregulated.