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Gene Information
Gene symbol: TNFRSF10C
Gene name: tumor necrosis factor receptor superfamily, member 10c, decoy without an intracellular domain
HGNC ID: 11906
Synonyms: DcR1, TRAILR3, LIT, TRID, CD263
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | BCL2A1 | 1 hits |
| 2 | CASP2 | 1 hits |
| 3 | CASP8 | 1 hits |
| 4 | CAST | 1 hits |
| 5 | CCL4 | 1 hits |
| 6 | CDK2 | 1 hits |
| 7 | CFLAR | 1 hits |
| 8 | GADD45B | 1 hits |
| 9 | IL1B | 1 hits |
| 10 | NFKB1 | 1 hits |
| 11 | NFKB2 | 1 hits |
| 12 | PIM2 | 1 hits |
| 13 | RELA | 1 hits |
| 14 | SOD2 | 1 hits |
| 15 | TNFAIP3 | 1 hits |
| 16 | TNFRSF10A | 1 hits |
| 17 | TNFRSF10B | 1 hits |
| 18 | TNFRSF10D | 1 hits |
| 19 | TNFSF10 | 1 hits |
| 20 | VEGFA | 1 hits |
| 21 | XIAP | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 10889508 | Involvement of the apoptosis-mediating CD95/CD95 ligand system in MS has been demonstrated. |
| 2 | 10889508 | Here, we report that (auto)antigen-specific human T cells are not killed in vitro by soluble TNF-related apoptosis-inducing ligand (TRAIL) although expressing death-inducing receptors, TRAIL receptor 1 (TRAIL-R1) and TRAIL-R2. |
| 3 | 10889508 | The (auto)antigen-specific T cells were also resistant to specific TRAIL-R1/TRAIL-R2-directed induction of apoptosis, indicating that coexpression of the truncated TRAIL-R3 and TRAIL-R4 in these T cells is not responsible for the observed resistance. |
| 4 | 10889508 | In contrast to CD95, the role of TRAIL receptors in MS might not involve regulation of T cell vulnerability. |
| 5 | 22986450 | Specific targets in this category included genes asso-ciated with the intrinsic and extrinsic apoptotic pathways (CFLAR, TNFAIP3, TNFRSF10D, SOD2, BCL2A1, BIRC4, PIM2, TNFSF10, TNFRSF10C, CASP2 and CASP8) and genes that act via the NFĸB pathway and other mechanisms to prolong cell viability (NFKB1, NFKB2 and RELA, IL1B, CAST, CDK2,GADD45B, BCL3, BIRC3, CDK2, IL1A, PBEF1, IL6, CXCL1, CCL4 and VEGF). |
| 6 | 22986450 | Moreover, we demonstrate that the X-linked inhibitor of apoptosis protein remained abundant in polymorphonuclear leukocytes over 48 h of LVS infection, whereas BAX mRNA and protein were progressively downregulated. |