Ignet

Gene Information

Gene symbol: ZNRD1

Gene name: zinc ribbon domain containing 1

HGNC ID: 13182

Synonyms: hZR14, HTEX-6, tctex-6, RPA12

Related Genes

#	Gene Symbol	Number of hits
1	HCP5	1 hits
2	HLA-B	1 hits
3	HLA-C	1 hits

Related Sentences

#	PMID	Sentence
1	18982067	HIV-1 disease-influencing effects associated with ZNRD1, HCP5 and HLA-C alleles are attributable mainly to either HLA-A10 or HLA-B*57 alleles.
2	18982067	A recent genome-wide association study (GWAS) suggested that polymorphisms in or around the genes HCP5, HLA-C and ZNRD1 confer restriction against HIV-1 viral replication or disease progression.
3	18982067	We also find that because of linkage disequilibrium (LD) patterns, the dominant viral load- and disease-influencing associations for the ZNRD1 or HLA-C and HCP5 alleles are apparent mainly when these alleles are present in HLA-A10- or HLA-B*57-containing haplotypes, respectively.
4	18982067	Furthermore, HCP5 and HLA-C alleles stratify B57-containing genotypes into those that associate with either striking disease retardation or progressive disease, providing one explanation for the long-standing conundrum of why some HLA-B57-carrying individuals are long-term non-progressors, whereas others exhibit progressive disease.
5	18982067	They specifically demonstrate that the influence of ZNRD1 alleles on disease progression rates are attributable to HLA-A10, help clarify the relationship between the HCP5, HLA-C and HLA-B57 alleles, and reaffirm a critical role of HLA-B57 alleles in HIV disease.
6	18982067	HIV-1 disease-influencing effects associated with ZNRD1, HCP5 and HLA-C alleles are attributable mainly to either HLA-A10 or HLA-B*57 alleles.
7	18982067	A recent genome-wide association study (GWAS) suggested that polymorphisms in or around the genes HCP5, HLA-C and ZNRD1 confer restriction against HIV-1 viral replication or disease progression.
8	18982067	We also find that because of linkage disequilibrium (LD) patterns, the dominant viral load- and disease-influencing associations for the ZNRD1 or HLA-C and HCP5 alleles are apparent mainly when these alleles are present in HLA-A10- or HLA-B*57-containing haplotypes, respectively.
9	18982067	Furthermore, HCP5 and HLA-C alleles stratify B57-containing genotypes into those that associate with either striking disease retardation or progressive disease, providing one explanation for the long-standing conundrum of why some HLA-B57-carrying individuals are long-term non-progressors, whereas others exhibit progressive disease.
10	18982067	They specifically demonstrate that the influence of ZNRD1 alleles on disease progression rates are attributable to HLA-A10, help clarify the relationship between the HCP5, HLA-C and HLA-B57 alleles, and reaffirm a critical role of HLA-B57 alleles in HIV disease.
11	18982067	HIV-1 disease-influencing effects associated with ZNRD1, HCP5 and HLA-C alleles are attributable mainly to either HLA-A10 or HLA-B*57 alleles.
12	18982067	A recent genome-wide association study (GWAS) suggested that polymorphisms in or around the genes HCP5, HLA-C and ZNRD1 confer restriction against HIV-1 viral replication or disease progression.
13	18982067	We also find that because of linkage disequilibrium (LD) patterns, the dominant viral load- and disease-influencing associations for the ZNRD1 or HLA-C and HCP5 alleles are apparent mainly when these alleles are present in HLA-A10- or HLA-B*57-containing haplotypes, respectively.
14	18982067	Furthermore, HCP5 and HLA-C alleles stratify B57-containing genotypes into those that associate with either striking disease retardation or progressive disease, providing one explanation for the long-standing conundrum of why some HLA-B57-carrying individuals are long-term non-progressors, whereas others exhibit progressive disease.
15	18982067	They specifically demonstrate that the influence of ZNRD1 alleles on disease progression rates are attributable to HLA-A10, help clarify the relationship between the HCP5, HLA-C and HLA-B57 alleles, and reaffirm a critical role of HLA-B57 alleles in HIV disease.
16	18982067	HIV-1 disease-influencing effects associated with ZNRD1, HCP5 and HLA-C alleles are attributable mainly to either HLA-A10 or HLA-B*57 alleles.
17	18982067	A recent genome-wide association study (GWAS) suggested that polymorphisms in or around the genes HCP5, HLA-C and ZNRD1 confer restriction against HIV-1 viral replication or disease progression.
18	18982067	We also find that because of linkage disequilibrium (LD) patterns, the dominant viral load- and disease-influencing associations for the ZNRD1 or HLA-C and HCP5 alleles are apparent mainly when these alleles are present in HLA-A10- or HLA-B*57-containing haplotypes, respectively.
19	18982067	Furthermore, HCP5 and HLA-C alleles stratify B57-containing genotypes into those that associate with either striking disease retardation or progressive disease, providing one explanation for the long-standing conundrum of why some HLA-B57-carrying individuals are long-term non-progressors, whereas others exhibit progressive disease.
20	18982067	They specifically demonstrate that the influence of ZNRD1 alleles on disease progression rates are attributable to HLA-A10, help clarify the relationship between the HCP5, HLA-C and HLA-B57 alleles, and reaffirm a critical role of HLA-B57 alleles in HIV disease.