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Gene Information
Gene symbol: CDKN1B
Gene name: cyclin-dependent kinase inhibitor 1B (p27, Kip1)
HGNC ID: 1785
Synonyms: KIP1, P27KIP1
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | BCL2 | 1 hits |
| 2 | CCNA2 | 1 hits |
| 3 | CDK2 | 1 hits |
| 4 | CDK4 | 1 hits |
| 5 | CDK6 | 1 hits |
| 6 | CHKA | 1 hits |
| 7 | MKI67 | 1 hits |
| 8 | PSMD9 | 1 hits |
| 9 | SYNPO | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 9551393 | Immunoprecipitation experiments revealed that p27Kip1 protein associates with Cdk2 and Cdk4, but not with Cdk6. |
| 2 | 10526006 | Additionally, serial section analysis revealed that Ki-67-positive cells in the crescents were frequently cyclin-A positive and Bcl-2 positive, but seldom cyclin-B(1) positive. |
| 3 | 10526006 | Moreover, the expression of cyclin-dependent kinase inhibitor p27(Kip1) was low in the cellular crescents, despite being exclusively positive in podocytes within the same section. |
| 4 | 10526006 | We concluded that the major component of the cellular crescents is made up of PECs and that apparent expression of cyclins and Bcl-2 and restrained expression of p27(Kip1) may be synergistically associated with the development of cellular crescents in human CRGN. |
| 5 | 10526006 | Additionally, serial section analysis revealed that Ki-67-positive cells in the crescents were frequently cyclin-A positive and Bcl-2 positive, but seldom cyclin-B(1) positive. |
| 6 | 10526006 | Moreover, the expression of cyclin-dependent kinase inhibitor p27(Kip1) was low in the cellular crescents, despite being exclusively positive in podocytes within the same section. |
| 7 | 10526006 | We concluded that the major component of the cellular crescents is made up of PECs and that apparent expression of cyclins and Bcl-2 and restrained expression of p27(Kip1) may be synergistically associated with the development of cellular crescents in human CRGN. |
| 8 | 10879738 | All of the cellular lesions expressed cytokeratin, frequently with Ki-67 (82.4%) and less frequently with cyclin A (17.7%), but were invariably negative for podocyte markers (PHM-5 and synaptopodin) and CKIs (p27kip1 and p57kip2). |
| 9 | 10879738 | In conclusion, proliferation of PEC, sustained by repression of CKIs in nature and simultaneous activation of cyclin A, is the actual molecular background to the cellular lesions in FGS. |