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Gene Information
Gene symbol: APOD
Gene name: apolipoprotein D
HGNC ID: 612
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | C4A | 1 hits |
| 2 | FASN | 1 hits |
| 3 | HSPA1A | 1 hits |
| 4 | INS | 1 hits |
| 5 | IRS1 | 1 hits |
| 6 | LPL | 1 hits |
| 7 | MAPK8 | 1 hits |
| 8 | MYH14 | 1 hits |
| 9 | PPARA | 1 hits |
| 10 | PPARG | 1 hits |
| 11 | RBP4 | 1 hits |
| 12 | UCP2 | 1 hits |
| 13 | UCP3 | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 1485944 | A number of candidate genes have been studied with the aim of demonstrating either association or linkage with the disease; in South Indians the only positive results thus far have been associations between non-insulin-dependent diabetes mellitus and the genes for insulin, apolipoprotein D and complement component C4B. |
| 2 | 7913935 | After a recently described association between the apolipoprotein-D (Apo-D) gene polymorphism and non-insulin-dependent diabetes mellitus by our group, we have now looked at a TaqI polymorphism of the Apo-D gene in two other components of syndrome X, namely obesity and hyperinsulinemia. |
| 3 | 7913935 | Furthermore, in the obese group an association was found between the Apo-D genotype and fasting insulin (P < 0.001). |
| 4 | 7913935 | After a recently described association between the apolipoprotein-D (Apo-D) gene polymorphism and non-insulin-dependent diabetes mellitus by our group, we have now looked at a TaqI polymorphism of the Apo-D gene in two other components of syndrome X, namely obesity and hyperinsulinemia. |
| 5 | 7913935 | Furthermore, in the obese group an association was found between the Apo-D genotype and fasting insulin (P < 0.001). |
| 6 | 15369805 | One-hundred-forty-four genes were differentially expressed in myotubes from donors with type 2 diabetes compared with control subjects, including HSP70, apolipoprotein D/E, tropomyosin, myosin, and actin previously reported from in vivo studies of diabetic skeletal muscle. |
| 7 | 18548385 | We also observed changes in transcript abundance of PPAR-gamma, PPAR-alpha, FAS, LPL, UCP2, UCP3, CPT1, RxR, ObRb, ApoAII, ApoD, and IRS1 in liver, muscle, and adipose tissue, suggesting treatment-induced effects on these genes. |
| 8 | 19390610 | Stress and inflammatory signaling pathways--such as Jun-N-terminal Kinase (JNK) signaling--repress IIS, curtailing anabolic processes to promote stress tolerance and extend lifespan. |
| 9 | 19390610 | While this interaction constitutes an adaptive response that allows managing energy resources under stress conditions, excessive JNK activity in adipose tissue of vertebrates has been found to cause insulin resistance, promoting type II diabetes. |
| 10 | 19390610 | We show that JNK signaling is required for metabolic homeostasis in flies and that this function is mediated by the Drosophila Lipocalin family member Neural Lazarillo (NLaz), a homologue of vertebrate Apolipoprotein D (ApoD) and Retinol Binding Protein 4 (RBP4). |