# |
PMID |
Sentence |
1 |
15862893
|
Complex disruption of energy homeostasis in orexin neuron-deficient transgenic mice (orexin/ataxin-3 mice) is also manifested as late-onset obesity despite eating less.
|
2 |
16407534
|
Mice with ablation of MCH neurons were generated using toxin (ataxin-3)-mediated ablation strategy.
|
3 |
16407534
|
To study the role of MCH neurons on obesity secondary to leptin deficiency, we generated mice deficient in both ob gene product (leptin) and MCH neurons.
|
4 |
16407534
|
Absence of MCH neurons in ob/ob mice improved obesity, diabetes, and hepatic steatosis, suggesting that MCH neurons are important mediators of the response to leptin deficiency.
|
5 |
17000876
|
Regulation of retrotranslocation by p97-associated deubiquitinating enzyme ataxin-3.
|
6 |
17000876
|
We report that efficient elimination of misfolded ER proteins also involves ataxin-3 (atx3), a p97-associated deubiquitinating enzyme mutated in type-3 spinocerebellar ataxia.
|
7 |
17000876
|
Regulation of retrotranslocation by p97-associated deubiquitinating enzyme ataxin-3.
|
8 |
17000876
|
We report that efficient elimination of misfolded ER proteins also involves ataxin-3 (atx3), a p97-associated deubiquitinating enzyme mutated in type-3 spinocerebellar ataxia.
|