# |
PMID |
Sentence |
1 |
1647992
|
CR2-expressing RINm5F cells secreted 78-33% less insulin than parental cells or cells transduced with an antisense vector and could be infected with high-titer EBV.
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2 |
8142907
|
The sites on Fc epsilon RII/CD23 that are possibly involved in the interaction with IgE and with another ligand, CD21/CR2, are proposed.
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3 |
10433799
|
Contribution of the innate immune system to autoimmune diabetes: a role for the CR1/CR2 complement receptors.
|
4 |
10433799
|
The complement component of the innate immune system regulates B cell activation and tolerance through complement receptors CR1/CR2.
|
5 |
10433799
|
Examination of the lymphoid compartments of NOD mice revealed striking expansion of a splenic B cell subset with high cell surface expression of CR1/CR2.
|
6 |
10433799
|
Importantly, long-term in vivo blockade of C3 binding to CR1/CR2 prevented the emergence of the CR1/CR2(hi) B cells and afforded resistance to autoimmune diabetes in NOD mice.
|
7 |
10433799
|
Contribution of the innate immune system to autoimmune diabetes: a role for the CR1/CR2 complement receptors.
|
8 |
10433799
|
The complement component of the innate immune system regulates B cell activation and tolerance through complement receptors CR1/CR2.
|
9 |
10433799
|
Examination of the lymphoid compartments of NOD mice revealed striking expansion of a splenic B cell subset with high cell surface expression of CR1/CR2.
|
10 |
10433799
|
Importantly, long-term in vivo blockade of C3 binding to CR1/CR2 prevented the emergence of the CR1/CR2(hi) B cells and afforded resistance to autoimmune diabetes in NOD mice.
|
11 |
10433799
|
Contribution of the innate immune system to autoimmune diabetes: a role for the CR1/CR2 complement receptors.
|
12 |
10433799
|
The complement component of the innate immune system regulates B cell activation and tolerance through complement receptors CR1/CR2.
|
13 |
10433799
|
Examination of the lymphoid compartments of NOD mice revealed striking expansion of a splenic B cell subset with high cell surface expression of CR1/CR2.
|
14 |
10433799
|
Importantly, long-term in vivo blockade of C3 binding to CR1/CR2 prevented the emergence of the CR1/CR2(hi) B cells and afforded resistance to autoimmune diabetes in NOD mice.
|
15 |
10433799
|
Contribution of the innate immune system to autoimmune diabetes: a role for the CR1/CR2 complement receptors.
|
16 |
10433799
|
The complement component of the innate immune system regulates B cell activation and tolerance through complement receptors CR1/CR2.
|
17 |
10433799
|
Examination of the lymphoid compartments of NOD mice revealed striking expansion of a splenic B cell subset with high cell surface expression of CR1/CR2.
|
18 |
10433799
|
Importantly, long-term in vivo blockade of C3 binding to CR1/CR2 prevented the emergence of the CR1/CR2(hi) B cells and afforded resistance to autoimmune diabetes in NOD mice.
|