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Gene Information
Gene symbol: GSTM2
Gene name: glutathione S-transferase mu 2 (muscle)
HGNC ID: 4634
Synonyms: GST4
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | ANXA1 | 1 hits |
| 2 | APOA4 | 1 hits |
| 3 | GSTA1 | 1 hits |
| 4 | GSTCD | 1 hits |
| 5 | GSTK1 | 1 hits |
| 6 | GSTM4 | 1 hits |
| 7 | GSTP1 | 1 hits |
| 8 | GSTT1 | 1 hits |
| 9 | LPAL2 | 1 hits |
| 10 | MAFK | 1 hits |
| 11 | NFE2L2 | 1 hits |
| 12 | PRDX2 | 1 hits |
| 13 | SERPINC1 | 1 hits |
| 14 | SOD2 | 1 hits |
| 15 | SOD3 | 1 hits |
| 16 | VCL | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 23111281 | Gene expression profiles of BB rat islets were highly distinct from F344 islets and under-expressed numerous genes involved in ROS metabolism, including glutathione S-transferase (GST) family members (Gstm2, Gstm4, Gstm7, Gstt1, Gstp1, and Gstk1), superoxide dismutases (Sod2 and Sod3), peroxidases, and peroxiredoxins. |
| 2 | 23767859 | Expressions of glutathione S-transferases Mu (GSTM) and nuclear factor erythroid 2-related factor 2 (Nrf2) were detected by western blot, and apoptosis was analyzed using a flow cytometer. |
| 3 | 23767859 | In vitro, resveratrol inhibited the proliferation of mesangial cells caused by high glucose and down-regulated GSTM and Nrf2 expressions in a dose-dependent manner. |
| 4 | 23767859 | Expressions of glutathione S-transferases Mu (GSTM) and nuclear factor erythroid 2-related factor 2 (Nrf2) were detected by western blot, and apoptosis was analyzed using a flow cytometer. |
| 5 | 23767859 | In vitro, resveratrol inhibited the proliferation of mesangial cells caused by high glucose and down-regulated GSTM and Nrf2 expressions in a dose-dependent manner. |
| 6 | 23791845 | Diabetic patients showed increased VAT abundance of glutathione S-transferase Mu 2, peroxiredoxin-2, antithrombin-III, apolipoprotein A-IV, Ig κ chain C region, mitochondrial aldehyde dehydrogenase and actin, and decreased abundance of annexin-A1, retinaldehyde dehydrogenase-1, and vinculin, compared with their non-diabetic counterparts. |