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Gene Information

Gene symbol: ICAM2

Gene name: intercellular adhesion molecule 2

HGNC ID: 5345

Synonyms: CD102

Related Genes

# Gene Symbol Number of hits
1 BCL2 1 hits
2 CFLAR 1 hits
3 ICAM1 1 hits
4 ICAM3 1 hits
5 ITGAL 1 hits
6 SELE 1 hits
7 SELP 1 hits
8 SERPINB9 1 hits
9 VCAM1 1 hits

Related Sentences

# PMID Sentence
1 1340530 The blood monocytes adhere to endothelial cells unstimulated and after stimulation by interleukin-1, tumor necrosis factor or other mediators.
2 1340530 E-Selectin (ELAM-1), P-Selectin (GMP-140) and receptors of the immunoglobulin superfamily (ICAM-1, ICAM-2 and VCAM-1) are expressed on endothelial cells in basal conditions and after activation.
3 1340530 As estimated by flow cytometry CD11b/CD18 expression on diabetic monocytes was increased.
4 1340530 Pentoxifylline reduced CD11b/CD18 expression on normal and diabetic monocytes.
5 7836774 The intercellular adhesion molecule-3 (ICAM-3) has been identified as the third LFA-1 ligand in addition to ICAM-1 and ICAM-2.
6 15565861 Cells were transduced using a Maloney murine leukemia virus (MLV) vector coding for yellow fluorescent protein (YFP) and for one of the following antiapoptotic genes: cFLIP, FADD-DN, BcL-2, PI-9, and ICAM-2.
7 15565861 The data demonstrate that cFLIP, FADD-DN, and PI-9 are significantly more effective in protecting NIT-1 cells than BcL-2 and ICAM-2.
8 15565861 Additionally, the data show that despite its weak in vitro inhibition of caspase-3, PI-9 affords significant protection against TNF-alpha-induced apoptosis in these cells.
9 15565861 Cells were transduced using a Maloney murine leukemia virus (MLV) vector coding for yellow fluorescent protein (YFP) and for one of the following antiapoptotic genes: cFLIP, FADD-DN, BcL-2, PI-9, and ICAM-2.
10 15565861 The data demonstrate that cFLIP, FADD-DN, and PI-9 are significantly more effective in protecting NIT-1 cells than BcL-2 and ICAM-2.
11 15565861 Additionally, the data show that despite its weak in vitro inhibition of caspase-3, PI-9 affords significant protection against TNF-alpha-induced apoptosis in these cells.