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Gene Information

Gene symbol: OSMR

Gene name: oncostatin M receptor

HGNC ID: 8507

Synonyms: OSMRB

Related Genes

# Gene Symbol Number of hits
1 OSM 1 hits

Related Sentences

# PMID Sentence
1 16410783 OSM and its specific OSM receptor subunit beta (OSMRbeta) were induced upon injury.
2 16410783 OSM protein was localized in PMN in very early wounds, whereas OSMRbeta could be detected on macrophages, keratinocytes, and fibroblasts later in repair.
3 16410783 PMN-depleted wounds were characterized by a nearly complete loss of OSM but not OSMRbeta mRNA and protein expression within the initial 16-24 hours after injury.
4 16410783 Moreover, a leptin-mediated improvement of chronic wounds in ob/ob mice was paralleled by a complete inhibition of PMN influx associated again with a dramatic loss of OSM expression at the wound site.
5 16410783 OSM and its specific OSM receptor subunit beta (OSMRbeta) were induced upon injury.
6 16410783 OSM protein was localized in PMN in very early wounds, whereas OSMRbeta could be detected on macrophages, keratinocytes, and fibroblasts later in repair.
7 16410783 PMN-depleted wounds were characterized by a nearly complete loss of OSM but not OSMRbeta mRNA and protein expression within the initial 16-24 hours after injury.
8 16410783 Moreover, a leptin-mediated improvement of chronic wounds in ob/ob mice was paralleled by a complete inhibition of PMN influx associated again with a dramatic loss of OSM expression at the wound site.
9 16410783 OSM and its specific OSM receptor subunit beta (OSMRbeta) were induced upon injury.
10 16410783 OSM protein was localized in PMN in very early wounds, whereas OSMRbeta could be detected on macrophages, keratinocytes, and fibroblasts later in repair.
11 16410783 PMN-depleted wounds were characterized by a nearly complete loss of OSM but not OSMRbeta mRNA and protein expression within the initial 16-24 hours after injury.
12 16410783 Moreover, a leptin-mediated improvement of chronic wounds in ob/ob mice was paralleled by a complete inhibition of PMN influx associated again with a dramatic loss of OSM expression at the wound site.