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Gene Information
Gene symbol: PRKAB1
Gene name: protein kinase, AMP-activated, beta 1 non-catalytic subunit
HGNC ID: 9378
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | HOXC6 | 1 hits |
| 2 | INS | 1 hits |
| 3 | MAPK1 | 1 hits |
| 4 | MYO18B | 1 hits |
| 5 | PRKAA1 | 1 hits |
| 6 | PRKAA2 | 1 hits |
| 7 | PRKAB2 | 1 hits |
| 8 | PYGM | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 12747837 | AMP-activated protein kinase (AMPK) is a multisubstrate enzyme activated by increases in AMP during metabolic stress caused by exercise, hypoxia, lack of cell nutrients, as well as hormones, including adiponectin and leptin. |
| 2 | 12747837 | Mammalian AMPK is an alphabetagamma heterotrimer with multiple isoforms of each subunit comprising alpha1, alpha2, beta1, beta2, gamma1, gamma2, and gamma3, which have varying tissue and subcellular expression. |
| 3 | 12747837 | Overexpressed AMPK beta1 localized to specific mammalian subcellular structures that corresponded with the expression pattern of glycogen phosphorylase. |
| 4 | 16505254 | Haplotype structures and large-scale association testing of the 5' AMP-activated protein kinase genes PRKAA2, PRKAB1, and PRKAB2 [corrected] with type 2 diabetes. |
| 5 | 16505254 | Of the seven genes that encode AMPK isoforms, we initially chose PRKAA2, PRKAB1, and PRKAB2 because of their higher prior probability of association with type 2 diabetes, based on previous reports of genetic linkage, functional molecular studies, expression patterns, and pharmacological evidence. |
| 6 | 16505254 | Several nominal associations of variants in PRKAA2 and PRKAB1 with BMI appear to be consistent with statistical noise. |
| 7 | 16505254 | Haplotype structures and large-scale association testing of the 5' AMP-activated protein kinase genes PRKAA2, PRKAB1, and PRKAB2 [corrected] with type 2 diabetes. |
| 8 | 16505254 | Of the seven genes that encode AMPK isoforms, we initially chose PRKAA2, PRKAB1, and PRKAB2 because of their higher prior probability of association with type 2 diabetes, based on previous reports of genetic linkage, functional molecular studies, expression patterns, and pharmacological evidence. |
| 9 | 16505254 | Several nominal associations of variants in PRKAA2 and PRKAB1 with BMI appear to be consistent with statistical noise. |
| 10 | 16505254 | Haplotype structures and large-scale association testing of the 5' AMP-activated protein kinase genes PRKAA2, PRKAB1, and PRKAB2 [corrected] with type 2 diabetes. |
| 11 | 16505254 | Of the seven genes that encode AMPK isoforms, we initially chose PRKAA2, PRKAB1, and PRKAB2 because of their higher prior probability of association with type 2 diabetes, based on previous reports of genetic linkage, functional molecular studies, expression patterns, and pharmacological evidence. |
| 12 | 16505254 | Several nominal associations of variants in PRKAA2 and PRKAB1 with BMI appear to be consistent with statistical noise. |
| 13 | 16508085 | Crystallization of the glycogen-binding domain of the AMP-activated protein kinase beta subunit and preliminary X-ray analysis. |
| 14 | 23028138 | We found differential DNA methylation of genes in biological pathways including mitogen-activated protein kinase (MAPK), insulin, and calcium signaling (P ≤ 0.007) and of individual genes with known function in muscle, including MAPK1, MYO18B, HOXC6, and the AMP-activated protein kinase subunit PRKAB1 in skeletal muscle of FH(+) compared with FH(-) men. |
| 15 | 23028138 | DNA methylation of genes in retinol metabolism and calcium signaling pathways (P < 3 × 10(-6)) and with known functions in muscle and T2D including MEF2A, RUNX1, NDUFC2, and THADA decreased after exercise. |
| 16 | 23028138 | In addition, both expression and methylation of several genes, i.e., ADIPOR1, BDKRB2, and TRIB1, changed after exercise. |