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Gene Information

Gene symbol: SERTAD2

Gene name: SERTA domain containing 2

HGNC ID: 30784

Synonyms: TRIP-Br2, KIAA0127, Sei-2

Related Genes

# Gene Symbol Number of hits
1 INS 1 hits

Related Sentences

# PMID Sentence
1 23291629 Ablation of TRIP-Br2, a regulator of fat lipolysis, thermogenesis and oxidative metabolism, prevents diet-induced obesity and insulin resistance.
2 23291629 TRIP-Br2-null mice are resistant to obesity and obesity-related insulin resistance.
3 23291629 Adipocytes of these knockout mice showed greater stimulated lipolysis secondary to enhanced expression of hormone sensitive lipase (HSL) and β3-adrenergic (Adrb3) receptors.
4 23291629 These data, together with the observation that TRIP-Br2 expression is selectively elevated in visceral fat in obese humans, suggests that this transcriptional co-regulator is a new therapeutic target for counteracting the development of obesity, insulin resistance and hyperlipidemia.
5 23291629 Ablation of TRIP-Br2, a regulator of fat lipolysis, thermogenesis and oxidative metabolism, prevents diet-induced obesity and insulin resistance.
6 23291629 TRIP-Br2-null mice are resistant to obesity and obesity-related insulin resistance.
7 23291629 Adipocytes of these knockout mice showed greater stimulated lipolysis secondary to enhanced expression of hormone sensitive lipase (HSL) and β3-adrenergic (Adrb3) receptors.
8 23291629 These data, together with the observation that TRIP-Br2 expression is selectively elevated in visceral fat in obese humans, suggests that this transcriptional co-regulator is a new therapeutic target for counteracting the development of obesity, insulin resistance and hyperlipidemia.
9 23291629 Ablation of TRIP-Br2, a regulator of fat lipolysis, thermogenesis and oxidative metabolism, prevents diet-induced obesity and insulin resistance.
10 23291629 TRIP-Br2-null mice are resistant to obesity and obesity-related insulin resistance.
11 23291629 Adipocytes of these knockout mice showed greater stimulated lipolysis secondary to enhanced expression of hormone sensitive lipase (HSL) and β3-adrenergic (Adrb3) receptors.
12 23291629 These data, together with the observation that TRIP-Br2 expression is selectively elevated in visceral fat in obese humans, suggests that this transcriptional co-regulator is a new therapeutic target for counteracting the development of obesity, insulin resistance and hyperlipidemia.