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Gene Information
Gene symbol: SLC28A2
Gene name: solute carrier family 28 (sodium-coupled nucleoside transporter), member 2
HGNC ID: 11002
Synonyms: CNT2, SPNT1, HCNT2, HsT17153
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | INS | 1 hits |
| 2 | SLC28A1 | 1 hits |
| 3 | SLC28A3 | 1 hits |
| 4 | SLC29A1 | 1 hits |
| 5 | SLC29A2 | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 16014043 | Expression of concentrative nucleoside transporters SLC28 (CNT1, CNT2, and CNT3) along the rat nephron: effect of diabetes. |
| 2 | 16369729 | Isolated rat cardiomyocytes displayed the presence of detectable amounts of mRNA for ENT1, ENT2, CNT1, and CNT2. |
| 3 | 16369729 | The expression level of equilibrative transporters (ENT1, ENT2) decreased and of concentrative transporters (CNT1, CNT2) increased in myocytes isolated from diabetic rat. |
| 4 | 16369729 | The activity of ecto-5'-nucleotidase increased 2-fold in diabetic cells resulting in a rise of the activity ratio of ecto-5'-nucleotidase/adenosine deaminase from 28 to 56.These results indicate that in rat cardiomyocytes diabetes alters activities of adenosine metabolizing enzymes in such a way that conversion of AMP to IMP is favored in the cytosolic compartment, whereas the capability to produce adenosine extracellularly is increased. |
| 5 | 16873415 | Decrease of the insulin level below 10(-11) m resulted in over 3-fold increase in the nucleoside transporter CNT2 mRNA content. |
| 6 | 21266914 | In endothelial cells, 60%, 10%, and 30% of adenosine transport are mediated by ENT-1, ENT-2, and CNT-2, respectively. |
| 7 | 21266914 | It has been speculated that the increase in the activities of ENT-1 and CNT-2 may reduce the availability of adenosine to adenosine receptors, thereby weakening the vascular functions of adenosine. |
| 8 | 21266914 | In endothelial cells, 60%, 10%, and 30% of adenosine transport are mediated by ENT-1, ENT-2, and CNT-2, respectively. |
| 9 | 21266914 | It has been speculated that the increase in the activities of ENT-1 and CNT-2 may reduce the availability of adenosine to adenosine receptors, thereby weakening the vascular functions of adenosine. |