Ignet

Gene Information

Gene symbol: TAS2R10

Gene name:

HGNC ID:

Related Genes

#	Gene Symbol	Number of hits
1	AKT1	1 hits
2	MIRN98	1 hits
3	TAS2R14	1 hits
4	TRA	1 hits

Related Sentences

#	PMID	Sentence
1	9409713	The beta2 thyroid hormone receptor (TR-beta2) is by far the most abundant isoform in the pituitary, although transcripts of TR-alpha and TR-beta1 genes have been reported in developmental and adult rat pituitary gland.
2	22012613	Upregulation of TRB2 induced by miR-98 in the early lesions of large artery of type-2 diabetic rat.
3	22012613	Next, studies using RAOEC cells showed that the TRB2 expression was inhibited by the treatment of miR-98.
4	22012613	Finally, we validated the changes in TRB2 by studying one of the TRB2's substrates, Akt, in animal models.
5	22012613	TRB2 plays important roles in the pathogenesis of diabetic-2 large artery complications at early stage, and these effects may be modulated by miR-98.
6	22012613	Thus, targeting TRB2 and miR-98 could be considered as novel therapeutic strategies for the early large artery deficits in diabetic-2.
7	22012613	Upregulation of TRB2 induced by miR-98 in the early lesions of large artery of type-2 diabetic rat.
8	22012613	Next, studies using RAOEC cells showed that the TRB2 expression was inhibited by the treatment of miR-98.
9	22012613	Finally, we validated the changes in TRB2 by studying one of the TRB2's substrates, Akt, in animal models.
10	22012613	TRB2 plays important roles in the pathogenesis of diabetic-2 large artery complications at early stage, and these effects may be modulated by miR-98.
11	22012613	Thus, targeting TRB2 and miR-98 could be considered as novel therapeutic strategies for the early large artery deficits in diabetic-2.
12	22012613	Upregulation of TRB2 induced by miR-98 in the early lesions of large artery of type-2 diabetic rat.
13	22012613	Next, studies using RAOEC cells showed that the TRB2 expression was inhibited by the treatment of miR-98.
14	22012613	Finally, we validated the changes in TRB2 by studying one of the TRB2's substrates, Akt, in animal models.
15	22012613	TRB2 plays important roles in the pathogenesis of diabetic-2 large artery complications at early stage, and these effects may be modulated by miR-98.
16	22012613	Thus, targeting TRB2 and miR-98 could be considered as novel therapeutic strategies for the early large artery deficits in diabetic-2.
17	22012613	Upregulation of TRB2 induced by miR-98 in the early lesions of large artery of type-2 diabetic rat.
18	22012613	Next, studies using RAOEC cells showed that the TRB2 expression was inhibited by the treatment of miR-98.
19	22012613	Finally, we validated the changes in TRB2 by studying one of the TRB2's substrates, Akt, in animal models.
20	22012613	TRB2 plays important roles in the pathogenesis of diabetic-2 large artery complications at early stage, and these effects may be modulated by miR-98.
21	22012613	Thus, targeting TRB2 and miR-98 could be considered as novel therapeutic strategies for the early large artery deficits in diabetic-2.
22	22012613	Upregulation of TRB2 induced by miR-98 in the early lesions of large artery of type-2 diabetic rat.
23	22012613	Next, studies using RAOEC cells showed that the TRB2 expression was inhibited by the treatment of miR-98.
24	22012613	Finally, we validated the changes in TRB2 by studying one of the TRB2's substrates, Akt, in animal models.
25	22012613	TRB2 plays important roles in the pathogenesis of diabetic-2 large artery complications at early stage, and these effects may be modulated by miR-98.
26	22012613	Thus, targeting TRB2 and miR-98 could be considered as novel therapeutic strategies for the early large artery deficits in diabetic-2.