# |
PMID |
Sentence |
1 |
18708940
|
Subcutaneous panniculitis-like T-cell lymphoma (SPTL) is defined as a rare cytotoxic alpha/beta T-cell lymphoma characterized by primary involvement of subcutaneous tissue mimicking panniculitis and a predominant CD3+/CD4-/CD8+ phenotype in 2005 World Health Organization-European Organization for Research and Treatment of Cancer (WHO-EORTC) classification for cutaneous lymphomas.
|
2 |
18708940
|
Besides the common features described before, unusual CD4+/CD8- and CD4-/CD8- T-cell phenotypes were noted in 2 of our cases, respectively.
|
3 |
18708940
|
The frequency of rearranged TCRB, TCRG, and TCRD genes detected by BIOMED-2 multiplex polymerase chain reaction tubes was 80%, 67%, and 13%, respectively, with a total clonality detection rate of 100%.
|
4 |
18708940
|
We conclude that SPTL as a cytotoxic lymphoma derived from alpha/beta T cell has a predominant CD4-/CD8+ phenotype, but unusual CD4+/CD8- and CD4-/CD8- phenotypes do exist.
|
5 |
23352833
|
Autocrine CCL2, CXCL4, CXCL9 and CXCL10 signal in retinal endothelial cells and are enhanced in diabetic retinopathy.
|
6 |
23352833
|
This study aimed at examining the presence and role of chemokines (angiogenic CCL2/MCP-1 and angiostatic CXCL4/PF-4, CXCL9/Mig, CXCL10/IP-10) in proliferative diabetic retinopathy (PDR).
|
7 |
23352833
|
MCP-1, PF-4, Mig, IP-10 and VEGF levels in vitreous fluid from PDR patients were significantly higher than in controls.
|
8 |
23352833
|
Exploratory regression analysis identified associations between higher levels of IP-10 and inactive PDR and PDR with TRD.
|
9 |
23352833
|
VEGF levels correlated positively with MCP-1 and IP-10.
|
10 |
23352833
|
Significant positive correlations were observed between MCP-1 and IP-10 levels.
|
11 |
23352833
|
HRMEC produced MCP-1, Mig and IP-10 after stimulation with IFN-γ, IL-1β or lipopolysaccharide.
|
12 |
23352833
|
IFN-γ synergistically enhanced Mig and IP-10 production in response to IL-1β or lipopolysaccharide.
|
13 |
23352833
|
MCP-1 was produced by HRMEC in response to VEGF treatment and activated HRMEC via the ERK and Akt/PKB pathway.
|
14 |
23352833
|
On the other hand, phosphorylation of ERK induced by VEGF and MCP-1 was inhibited by PF-4, Mig and IP-10.
|