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Gene Information

Gene symbol: VASH1

Gene name: vasohibin 1

HGNC ID: 19964

Related Genes

# Gene Symbol Number of hits
1 ANXA2 1 hits
2 CCRK 1 hits
3 VEGFA 1 hits

Related Sentences

# PMID Sentence
1 19057892 Vitreous levels of vasohibin-1 and vascular endothelial growth factor in patients with proliferative diabetic retinopathy.
2 20659423 Here, we analyzed how the expression of the two active anti-angiogenic VASH-1 isoforms p36 and p42 was altered during physiological and pathological muscle angio-adaptation.
3 20659423 Interestingly, expression patterns for p36 and p42 were different between plantaris and heart muscles.
4 20659423 Both p36 and p42 isoforms were increased, a signal in favor of some vessel destabilization and regression.
5 20659423 In conclusion, our results bring evidence that endogenous VASH-1 isoforms p36 and p42 are key actors of physiological and pathological muscle angio-adaptation.
6 20659423 Here, we analyzed how the expression of the two active anti-angiogenic VASH-1 isoforms p36 and p42 was altered during physiological and pathological muscle angio-adaptation.
7 20659423 Interestingly, expression patterns for p36 and p42 were different between plantaris and heart muscles.
8 20659423 Both p36 and p42 isoforms were increased, a signal in favor of some vessel destabilization and regression.
9 20659423 In conclusion, our results bring evidence that endogenous VASH-1 isoforms p36 and p42 are key actors of physiological and pathological muscle angio-adaptation.
10 21228103 An increase in the renal levels of VEGF-A, VEGFR-2, transforming growth factor (TGF)-β1, and monocyte chemoattractant protein-1 in diabetic animals was significantly suppressed by AdhVASH-1 (immunoblotting).
11 21228103 AdhVASH-1 treatment significantly recovered the loss and altered the distribution patterns of nephrin and zonula occludens (ZO)-1 and suppressed the increase in the number of fibroblast-specific protein-1 (FSP-1(+)) and desmin(+) podocytes in diabetic mice.
12 21228103 In vitro, recombinant human VASH-1 (rhVASH-1) dose dependently suppressed the upregulation of VEGF induced by high ambient glucose (25 mM) in cultured mouse podocytes.
13 21228103 In addition, rhVASH-1 significantly recovered the mRNA levels of nephrin and the protein levels of ZO-1 and P-cadherin and suppressed the increase in protein levels of desmin, FSP-1, Snail, and Slug in podocytes under high-glucose condition.