Ignet

Gene Information

Gene symbol: ARMET

Gene name: arginine-rich, mutated in early stage tumors

HGNC ID: 15461

Related Genes

#	Gene Symbol	Number of hits
1	DNAJB11	1 hits
2	RPL10	1 hits
3	SF1	1 hits
4	SRPK1	1 hits
5	VEGFA	1 hits
6	WT1	1 hits

Related Sentences

#	PMID	Sentence
1	30641926	Altered VEGF Splicing Isoform Balance in Tumor Endothelium Involves Activation of Splicing Factors Srpk1 and Srsf1 by the Wilms' Tumor Suppressor Wt1.
2	30641926	In podocytes, the Wilms' tumor suppressor 1 (WT1) suppresses the Serine/arginine-rich protein-specific splicing factor kinase (SRPK1), and indirectly Serine/arginine-rich splicing factor 1 (Srsf1) activity, and alters VEGF splicing.
3	30641926	We analyzed VEGF isoforms, Wt1, Srpk1, and Srsf1 in normal and tumor endothelium.
4	30641926	Wt1, Srpk1, Srsf1, and the angiogenic VEGF164a isoform were highly expressed in tumor endothelium compared to normal lung endothelium.
5	30641926	Inducible conditional vessel-specific knockout of Wt1 reduced Wt1, Srpk1, and Srsf1 expression in endothelial cells and induced a shift towards the antiangiogenic VEGF120 isoform.
6	30641926	Wt1(-KTS) directly binds and activates both the promoters of Srpk1 and Srsf1 in endothelial cells.
7	30641926	In conclusion, Wt1 activates Srpk1 and Srsf1 and induces expression of angiogenic VEGF isoforms in tumor endothelium.
8	32203149	In cultured podocytes, ER stress increased ERdj3 and MANF intracellularly and in culture medium, whereas GRP94 (KDEL chaperone) increased only intracellularly.
9	32203149	ERdj3 and MANF secretion was blocked by the secretory trafficking inhibitor, brefeldin A.
10	32203149	Urinary ERdj3 and MANF increased in rats injected with tunicamycin (in the absence of proteinuria).
11	32203149	After induction of passive Heymann nephritis (PHN) and puromycin aminonucleoside nephrosis (PAN), there was an increase in glomerular ER stress, and appearance of ERdj3 and MANF in the urine, coinciding with the onset of proteinuria.
12	32203149	In conclusion, urinary ERdj3 and MANF reflect glomerular ER stress. 4-PBA protected against complement-mediated podocyte injury and the therapeutic response could be monitored by urinary ERdj3 and MANF.
13	32203149	In cultured podocytes, ER stress increased ERdj3 and MANF intracellularly and in culture medium, whereas GRP94 (KDEL chaperone) increased only intracellularly.
14	32203149	ERdj3 and MANF secretion was blocked by the secretory trafficking inhibitor, brefeldin A.
15	32203149	Urinary ERdj3 and MANF increased in rats injected with tunicamycin (in the absence of proteinuria).
16	32203149	After induction of passive Heymann nephritis (PHN) and puromycin aminonucleoside nephrosis (PAN), there was an increase in glomerular ER stress, and appearance of ERdj3 and MANF in the urine, coinciding with the onset of proteinuria.
17	32203149	In conclusion, urinary ERdj3 and MANF reflect glomerular ER stress. 4-PBA protected against complement-mediated podocyte injury and the therapeutic response could be monitored by urinary ERdj3 and MANF.
18	32203149	In cultured podocytes, ER stress increased ERdj3 and MANF intracellularly and in culture medium, whereas GRP94 (KDEL chaperone) increased only intracellularly.
19	32203149	ERdj3 and MANF secretion was blocked by the secretory trafficking inhibitor, brefeldin A.
20	32203149	Urinary ERdj3 and MANF increased in rats injected with tunicamycin (in the absence of proteinuria).
21	32203149	After induction of passive Heymann nephritis (PHN) and puromycin aminonucleoside nephrosis (PAN), there was an increase in glomerular ER stress, and appearance of ERdj3 and MANF in the urine, coinciding with the onset of proteinuria.
22	32203149	In conclusion, urinary ERdj3 and MANF reflect glomerular ER stress. 4-PBA protected against complement-mediated podocyte injury and the therapeutic response could be monitored by urinary ERdj3 and MANF.
23	32203149	In cultured podocytes, ER stress increased ERdj3 and MANF intracellularly and in culture medium, whereas GRP94 (KDEL chaperone) increased only intracellularly.
24	32203149	ERdj3 and MANF secretion was blocked by the secretory trafficking inhibitor, brefeldin A.
25	32203149	Urinary ERdj3 and MANF increased in rats injected with tunicamycin (in the absence of proteinuria).
26	32203149	After induction of passive Heymann nephritis (PHN) and puromycin aminonucleoside nephrosis (PAN), there was an increase in glomerular ER stress, and appearance of ERdj3 and MANF in the urine, coinciding with the onset of proteinuria.
27	32203149	In conclusion, urinary ERdj3 and MANF reflect glomerular ER stress. 4-PBA protected against complement-mediated podocyte injury and the therapeutic response could be monitored by urinary ERdj3 and MANF.
28	32203149	In cultured podocytes, ER stress increased ERdj3 and MANF intracellularly and in culture medium, whereas GRP94 (KDEL chaperone) increased only intracellularly.
29	32203149	ERdj3 and MANF secretion was blocked by the secretory trafficking inhibitor, brefeldin A.
30	32203149	Urinary ERdj3 and MANF increased in rats injected with tunicamycin (in the absence of proteinuria).
31	32203149	After induction of passive Heymann nephritis (PHN) and puromycin aminonucleoside nephrosis (PAN), there was an increase in glomerular ER stress, and appearance of ERdj3 and MANF in the urine, coinciding with the onset of proteinuria.
32	32203149	In conclusion, urinary ERdj3 and MANF reflect glomerular ER stress. 4-PBA protected against complement-mediated podocyte injury and the therapeutic response could be monitored by urinary ERdj3 and MANF.