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PMID |
Sentence |
1 |
34746448
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GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression.
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2 |
34746448
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We found that the decreased expression of nephrin, the cell marker of the podocyte, was significantly recovered with GAP-43 overexpression.
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3 |
34746448
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Moreover, overexpression of GAP-43 attenuated podocyte apoptosis by up-regulating the ratio of Bcl-2/Bax with LPS treatment.
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4 |
34746448
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Finally, Plaue and Rcan1 which are downstream target gene of NFATc1 decreased with overexpression of GAP-43 podocytes.
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5 |
34746448
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We concluded that GAP-43 attenuated podocyte injury by inhibiting calcineurin/NFATc1 signaling.
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6 |
34746448
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GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression.
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7 |
34746448
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We found that the decreased expression of nephrin, the cell marker of the podocyte, was significantly recovered with GAP-43 overexpression.
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8 |
34746448
|
Moreover, overexpression of GAP-43 attenuated podocyte apoptosis by up-regulating the ratio of Bcl-2/Bax with LPS treatment.
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9 |
34746448
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Finally, Plaue and Rcan1 which are downstream target gene of NFATc1 decreased with overexpression of GAP-43 podocytes.
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10 |
34746448
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We concluded that GAP-43 attenuated podocyte injury by inhibiting calcineurin/NFATc1 signaling.
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11 |
34746448
|
GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression.
|
12 |
34746448
|
We found that the decreased expression of nephrin, the cell marker of the podocyte, was significantly recovered with GAP-43 overexpression.
|
13 |
34746448
|
Moreover, overexpression of GAP-43 attenuated podocyte apoptosis by up-regulating the ratio of Bcl-2/Bax with LPS treatment.
|
14 |
34746448
|
Finally, Plaue and Rcan1 which are downstream target gene of NFATc1 decreased with overexpression of GAP-43 podocytes.
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15 |
34746448
|
We concluded that GAP-43 attenuated podocyte injury by inhibiting calcineurin/NFATc1 signaling.
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16 |
34746448
|
GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression.
|
17 |
34746448
|
We found that the decreased expression of nephrin, the cell marker of the podocyte, was significantly recovered with GAP-43 overexpression.
|
18 |
34746448
|
Moreover, overexpression of GAP-43 attenuated podocyte apoptosis by up-regulating the ratio of Bcl-2/Bax with LPS treatment.
|
19 |
34746448
|
Finally, Plaue and Rcan1 which are downstream target gene of NFATc1 decreased with overexpression of GAP-43 podocytes.
|
20 |
34746448
|
We concluded that GAP-43 attenuated podocyte injury by inhibiting calcineurin/NFATc1 signaling.
|
21 |
34746448
|
GAP-43 ameliorates Podocyte injury by decreasing nuclear NFATc1 expression.
|
22 |
34746448
|
We found that the decreased expression of nephrin, the cell marker of the podocyte, was significantly recovered with GAP-43 overexpression.
|
23 |
34746448
|
Moreover, overexpression of GAP-43 attenuated podocyte apoptosis by up-regulating the ratio of Bcl-2/Bax with LPS treatment.
|
24 |
34746448
|
Finally, Plaue and Rcan1 which are downstream target gene of NFATc1 decreased with overexpression of GAP-43 podocytes.
|
25 |
34746448
|
We concluded that GAP-43 attenuated podocyte injury by inhibiting calcineurin/NFATc1 signaling.
|