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Gene Information
Gene symbol: GPR137B
Gene name:
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | CLIC5 | 1 hits |
| 2 | INS | 1 hits |
| 3 | KHDRBS1 | 1 hits |
| 4 | NPHS1 | 1 hits |
| 5 | SLC2A4 | 1 hits |
| 6 | SQSTM1 | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 25168660 | Nucleobindin-2 is a positive regulator for insulin-stimulated glucose transporter 4 translocation in fenofibrate treated E11 podocytes. |
| 2 | 25168660 | To examine insulin stimulation of podocyte GLUT4 translocation, we established a protocol involving treatment with the PPARα agonist fenofibrate to induce E11 podocyte differentiation within 48 hours rather than 7-10 days, which is required for differentiation under the reported protocol. |
| 3 | 25168660 | Here we demonstrate that treatment with 200 μM fenofibrate for 36 hours following transfection had a dramatic effect on podocyte morphology, induced several podocyte specific protein expression markers (G protein-coupled receptor 137B, chloride intracellular channel 5, and nephrin) and resulted in insulin-stimulated GLUT4 translocation. |
| 4 | 25168660 | In addition, Nucleobindin-2 was found to constitutively associate with Septin 7 (the repressor of GLUT4 translocation), and knockdown of Nucleobindin-2 was found to completely abrogate insulin-stimulated GLUT4 translocation. |
| 5 | 25168660 | Together, these data suggest that Nucleobindin-2 may repress Septin7-induced inhibition of insulin-stimulated GLUT4 translocation in podocytes. |
| 6 | 32482387 | Interestingly, we detected an increase in LC3BII and SQSTM1/P62 in MPC5 through inhibiting TM7SF1, and which can be completely corrected after blocking the autolysosome degradation with chloroquine (CQ). |
| 7 | 32482387 | Moreover, inhibition of TM7SF1 expression did not increase the mRNA level of SQSTM1/P62. |
| 8 | 32482387 | Theses results suggested that inhibition of TM7SF1 led to impaired degradation of autophagy products, which manifest as an abnormal accumulation of LC3BII and SQSTM1/P62. |
| 9 | 32482387 | Interestingly, we detected an increase in LC3BII and SQSTM1/P62 in MPC5 through inhibiting TM7SF1, and which can be completely corrected after blocking the autolysosome degradation with chloroquine (CQ). |
| 10 | 32482387 | Moreover, inhibition of TM7SF1 expression did not increase the mRNA level of SQSTM1/P62. |
| 11 | 32482387 | Theses results suggested that inhibition of TM7SF1 led to impaired degradation of autophagy products, which manifest as an abnormal accumulation of LC3BII and SQSTM1/P62. |
| 12 | 32482387 | Interestingly, we detected an increase in LC3BII and SQSTM1/P62 in MPC5 through inhibiting TM7SF1, and which can be completely corrected after blocking the autolysosome degradation with chloroquine (CQ). |
| 13 | 32482387 | Moreover, inhibition of TM7SF1 expression did not increase the mRNA level of SQSTM1/P62. |
| 14 | 32482387 | Theses results suggested that inhibition of TM7SF1 led to impaired degradation of autophagy products, which manifest as an abnormal accumulation of LC3BII and SQSTM1/P62. |