Ignet

Gene Information

Gene symbol: MIRN199B

Gene name: microRNA 199b

HGNC ID: 31573

Related Genes

#	Gene Symbol	Number of hits
1	AKT1	1 hits
2	MARCH8	1 hits
3	RGS10	1 hits

Related Sentences

#	PMID	Sentence
1	34737809	miR-199b-5p mediates adriamycin-induced podocyte apoptosis by inhibiting the expression of RGS10.
2	34737809	The results from the present study demonstrated that miR-199b-5p was upregulated and that regulator of G-protein signaling 10 (RGS10) was downregulated in ADR-stimulated podocytes.
3	34737809	Overexpression of miR-199b-5p could inhibit RGS10 expression and stimulate podocyte apoptosis, whereas miR-199b-5p knockdown restored the levels of RGS10 and ameliorated podocyte apoptosis in ADR-induced podocytes.
4	34737809	Furthermore, the effects of miR-199b-5p overexpression could be significantly reversed by RGS10 overexpression.
5	34737809	In addition, podocyte transfection of miR-199b-5p activated the AKT/mechanistic target of rapamycin (mTOR) signaling, which was blocked following RGS10 overexpression.
6	34737809	Taken together, the present study demonstrated that miR-199b-5p upregulation could promote podocyte apoptosis by inhibiting the expression of RGS10 through the activation of AKT/mTOR signaling.
7	34737809	miR-199b-5p mediates adriamycin-induced podocyte apoptosis by inhibiting the expression of RGS10.
8	34737809	The results from the present study demonstrated that miR-199b-5p was upregulated and that regulator of G-protein signaling 10 (RGS10) was downregulated in ADR-stimulated podocytes.
9	34737809	Overexpression of miR-199b-5p could inhibit RGS10 expression and stimulate podocyte apoptosis, whereas miR-199b-5p knockdown restored the levels of RGS10 and ameliorated podocyte apoptosis in ADR-induced podocytes.
10	34737809	Furthermore, the effects of miR-199b-5p overexpression could be significantly reversed by RGS10 overexpression.
11	34737809	In addition, podocyte transfection of miR-199b-5p activated the AKT/mechanistic target of rapamycin (mTOR) signaling, which was blocked following RGS10 overexpression.
12	34737809	Taken together, the present study demonstrated that miR-199b-5p upregulation could promote podocyte apoptosis by inhibiting the expression of RGS10 through the activation of AKT/mTOR signaling.
13	34737809	miR-199b-5p mediates adriamycin-induced podocyte apoptosis by inhibiting the expression of RGS10.
14	34737809	The results from the present study demonstrated that miR-199b-5p was upregulated and that regulator of G-protein signaling 10 (RGS10) was downregulated in ADR-stimulated podocytes.
15	34737809	Overexpression of miR-199b-5p could inhibit RGS10 expression and stimulate podocyte apoptosis, whereas miR-199b-5p knockdown restored the levels of RGS10 and ameliorated podocyte apoptosis in ADR-induced podocytes.
16	34737809	Furthermore, the effects of miR-199b-5p overexpression could be significantly reversed by RGS10 overexpression.
17	34737809	In addition, podocyte transfection of miR-199b-5p activated the AKT/mechanistic target of rapamycin (mTOR) signaling, which was blocked following RGS10 overexpression.
18	34737809	Taken together, the present study demonstrated that miR-199b-5p upregulation could promote podocyte apoptosis by inhibiting the expression of RGS10 through the activation of AKT/mTOR signaling.
19	34737809	miR-199b-5p mediates adriamycin-induced podocyte apoptosis by inhibiting the expression of RGS10.
20	34737809	The results from the present study demonstrated that miR-199b-5p was upregulated and that regulator of G-protein signaling 10 (RGS10) was downregulated in ADR-stimulated podocytes.
21	34737809	Overexpression of miR-199b-5p could inhibit RGS10 expression and stimulate podocyte apoptosis, whereas miR-199b-5p knockdown restored the levels of RGS10 and ameliorated podocyte apoptosis in ADR-induced podocytes.
22	34737809	Furthermore, the effects of miR-199b-5p overexpression could be significantly reversed by RGS10 overexpression.
23	34737809	In addition, podocyte transfection of miR-199b-5p activated the AKT/mechanistic target of rapamycin (mTOR) signaling, which was blocked following RGS10 overexpression.
24	34737809	Taken together, the present study demonstrated that miR-199b-5p upregulation could promote podocyte apoptosis by inhibiting the expression of RGS10 through the activation of AKT/mTOR signaling.
25	34737809	miR-199b-5p mediates adriamycin-induced podocyte apoptosis by inhibiting the expression of RGS10.
26	34737809	The results from the present study demonstrated that miR-199b-5p was upregulated and that regulator of G-protein signaling 10 (RGS10) was downregulated in ADR-stimulated podocytes.
27	34737809	Overexpression of miR-199b-5p could inhibit RGS10 expression and stimulate podocyte apoptosis, whereas miR-199b-5p knockdown restored the levels of RGS10 and ameliorated podocyte apoptosis in ADR-induced podocytes.
28	34737809	Furthermore, the effects of miR-199b-5p overexpression could be significantly reversed by RGS10 overexpression.
29	34737809	In addition, podocyte transfection of miR-199b-5p activated the AKT/mechanistic target of rapamycin (mTOR) signaling, which was blocked following RGS10 overexpression.
30	34737809	Taken together, the present study demonstrated that miR-199b-5p upregulation could promote podocyte apoptosis by inhibiting the expression of RGS10 through the activation of AKT/mTOR signaling.
31	34737809	miR-199b-5p mediates adriamycin-induced podocyte apoptosis by inhibiting the expression of RGS10.
32	34737809	The results from the present study demonstrated that miR-199b-5p was upregulated and that regulator of G-protein signaling 10 (RGS10) was downregulated in ADR-stimulated podocytes.
33	34737809	Overexpression of miR-199b-5p could inhibit RGS10 expression and stimulate podocyte apoptosis, whereas miR-199b-5p knockdown restored the levels of RGS10 and ameliorated podocyte apoptosis in ADR-induced podocytes.
34	34737809	Furthermore, the effects of miR-199b-5p overexpression could be significantly reversed by RGS10 overexpression.
35	34737809	In addition, podocyte transfection of miR-199b-5p activated the AKT/mechanistic target of rapamycin (mTOR) signaling, which was blocked following RGS10 overexpression.
36	34737809	Taken together, the present study demonstrated that miR-199b-5p upregulation could promote podocyte apoptosis by inhibiting the expression of RGS10 through the activation of AKT/mTOR signaling.