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Gene Information

Gene symbol: MMP3

Gene name: matrix metallopeptidase 3 (stromelysin 1, progelatinase)

HGNC ID: 7173

Related Genes

#	Gene Symbol	Number of hits
1	COL1A1	1 hits
2	IL6	1 hits
3	MMP2	1 hits
4	NOS2A	1 hits
5	TIMP1	1 hits

Related Sentences

#	PMID	Sentence
1	19710627	There was robust induction of interleukin (IL)-6, along with MMP-3, -9, -10, and -14, but not MMP-2 or -12 by increased strain.
2	19710627	There was robust induction of interleukin (IL)-6, along with MMP-3, -9, -10, and -14, but not MMP-2 or -12 by increased strain.
3	19710627	There was robust induction of interleukin (IL)-6, along with MMP-3, -9, -10, and -14, but not MMP-2 or -12 by increased strain.
4	19710627	Neutralizing antibodies against IL-6 attenuated the strain-mediated induction of MMP-3 and -10.
5	19710627	Neutralizing antibodies against IL-6 attenuated the strain-mediated induction of MMP-3 and -10.
6	19710627	Neutralizing antibodies against IL-6 attenuated the strain-mediated induction of MMP-3 and -10.
7	19710627	Alport mice treated with a general inhibitor of nitric oxide synthase (L-NAME) developed significant hypertension and increased IL-6 and MMP-3 and -10 in their glomeruli relative to those of normotensive Alport mice.
8	19710627	Alport mice treated with a general inhibitor of nitric oxide synthase (L-NAME) developed significant hypertension and increased IL-6 and MMP-3 and -10 in their glomeruli relative to those of normotensive Alport mice.
9	19710627	Alport mice treated with a general inhibitor of nitric oxide synthase (L-NAME) developed significant hypertension and increased IL-6 and MMP-3 and -10 in their glomeruli relative to those of normotensive Alport mice.
10	12845621	These include increased synthesis of type IV collagen following hyperglycaemia-induced alteration of the pattern of podocyte-integrin expression, decreased expression of matrix metalloproteinases (MMP-2 and 3), and increased expression of tissue inhibitor of metalloproteinase (TIMP).
11	12845621	Other factors which may contribute to renal matrix accumulation include vascular endothelial growth factor (VEGF), since treatment with anti-VEGF antibodies attenuates glomerular basement membrane thickening, platelet-derived growth factor (PDGF) (B chain) and its receptor, which appear to be highly expressed in mesangial and visceral epithelial cells and might play a role in the development of diabetic nephropathy.