Ignet

Gene Information

Gene symbol: PHLDA1

Gene name: pleckstrin homology-like domain, family A, member 1

HGNC ID: 8933

Synonyms: TDAG51, DT1P1B11, PHRIP

Related Genes

#	Gene Symbol	Number of hits
1	AKT1	1 hits
2	NFE2L2	1 hits

Related Sentences

#	PMID	Sentence
1	35175494	TDAG51-Deficiency Podocytes are Protected from High-Glucose-Induced Damage Through Nrf2 Activation via the AKT-GSK-3β Pathway.
2	35175494	Mechanically, the inhibition of TDAG51 was capable of enhancing the activation of nuclear factor E2-related factor 2 (Nrf2) associated with the upregulation of AKT-glycogen synthase kinase-3β (GSK-3β) pathway.
3	35175494	The reduction of AKT abolished the activation of Nrf2 elicited by TDAG51 deficiency.
4	35175494	Additionally, the reduction of Nrf2 diminished the anti-HG injury effect elicited by TDAG51 deficiency.
5	35175494	Overall, these data demonstrate that TDAG51 deficiency defends against HG-induced podocyte damage through Nrf2 activation by regulating AKT-GSK-3β pathway.
6	35175494	TDAG51-Deficiency Podocytes are Protected from High-Glucose-Induced Damage Through Nrf2 Activation via the AKT-GSK-3β Pathway.
7	35175494	Mechanically, the inhibition of TDAG51 was capable of enhancing the activation of nuclear factor E2-related factor 2 (Nrf2) associated with the upregulation of AKT-glycogen synthase kinase-3β (GSK-3β) pathway.
8	35175494	The reduction of AKT abolished the activation of Nrf2 elicited by TDAG51 deficiency.
9	35175494	Additionally, the reduction of Nrf2 diminished the anti-HG injury effect elicited by TDAG51 deficiency.
10	35175494	Overall, these data demonstrate that TDAG51 deficiency defends against HG-induced podocyte damage through Nrf2 activation by regulating AKT-GSK-3β pathway.
11	35175494	TDAG51-Deficiency Podocytes are Protected from High-Glucose-Induced Damage Through Nrf2 Activation via the AKT-GSK-3β Pathway.
12	35175494	Mechanically, the inhibition of TDAG51 was capable of enhancing the activation of nuclear factor E2-related factor 2 (Nrf2) associated with the upregulation of AKT-glycogen synthase kinase-3β (GSK-3β) pathway.
13	35175494	The reduction of AKT abolished the activation of Nrf2 elicited by TDAG51 deficiency.
14	35175494	Additionally, the reduction of Nrf2 diminished the anti-HG injury effect elicited by TDAG51 deficiency.
15	35175494	Overall, these data demonstrate that TDAG51 deficiency defends against HG-induced podocyte damage through Nrf2 activation by regulating AKT-GSK-3β pathway.
16	35175494	TDAG51-Deficiency Podocytes are Protected from High-Glucose-Induced Damage Through Nrf2 Activation via the AKT-GSK-3β Pathway.
17	35175494	Mechanically, the inhibition of TDAG51 was capable of enhancing the activation of nuclear factor E2-related factor 2 (Nrf2) associated with the upregulation of AKT-glycogen synthase kinase-3β (GSK-3β) pathway.
18	35175494	The reduction of AKT abolished the activation of Nrf2 elicited by TDAG51 deficiency.
19	35175494	Additionally, the reduction of Nrf2 diminished the anti-HG injury effect elicited by TDAG51 deficiency.
20	35175494	Overall, these data demonstrate that TDAG51 deficiency defends against HG-induced podocyte damage through Nrf2 activation by regulating AKT-GSK-3β pathway.
21	35175494	TDAG51-Deficiency Podocytes are Protected from High-Glucose-Induced Damage Through Nrf2 Activation via the AKT-GSK-3β Pathway.
22	35175494	Mechanically, the inhibition of TDAG51 was capable of enhancing the activation of nuclear factor E2-related factor 2 (Nrf2) associated with the upregulation of AKT-glycogen synthase kinase-3β (GSK-3β) pathway.
23	35175494	The reduction of AKT abolished the activation of Nrf2 elicited by TDAG51 deficiency.
24	35175494	Additionally, the reduction of Nrf2 diminished the anti-HG injury effect elicited by TDAG51 deficiency.
25	35175494	Overall, these data demonstrate that TDAG51 deficiency defends against HG-induced podocyte damage through Nrf2 activation by regulating AKT-GSK-3β pathway.