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Gene Information
Gene symbol: SEMA3B
Gene name: sema domain, immunoglobulin domain (Ig), short basic domain, secreted, (semaphorin) 3B
HGNC ID: 10724
Synonyms: SemA, semaV, LUCA-1, sema5
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | AKT1 | 1 hits |
| 2 | CD2 | 1 hits |
| 3 | CD2AP | 1 hits |
| 4 | NELL1 | 1 hits |
| 5 | NPHS1 | 1 hits |
| 6 | NPHS2 | 1 hits |
| 7 | PLXNA1 | 1 hits |
| 8 | PLXNA2 | 1 hits |
| 9 | RORC | 1 hits |
| 10 | THSD7A | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 16541019 | Sema 3A decreased semaphorin 3B, plexin A1, A3, and D1 >/=50% and reduced plexin A2 mRNA to undetectable levels. |
| 2 | 16541019 | Sema 3A induced a dose-response podocin downregulation and decreased its interaction with CD2-associated protein and nephrin, as determined by Western analysis and co-immunoprecipitation. |
| 3 | 16541019 | Sema 3A induced a 10-fold increase in podocyte apoptosis and significantly decreased the activity of the Akt survival pathway. |
| 4 | 16541019 | Sema 3A decreased semaphorin 3B, plexin A1, A3, and D1 >/=50% and reduced plexin A2 mRNA to undetectable levels. |
| 5 | 16541019 | Sema 3A induced a dose-response podocin downregulation and decreased its interaction with CD2-associated protein and nephrin, as determined by Western analysis and co-immunoprecipitation. |
| 6 | 16541019 | Sema 3A induced a 10-fold increase in podocyte apoptosis and significantly decreased the activity of the Akt survival pathway. |
| 7 | 16541019 | Sema 3A decreased semaphorin 3B, plexin A1, A3, and D1 >/=50% and reduced plexin A2 mRNA to undetectable levels. |
| 8 | 16541019 | Sema 3A induced a dose-response podocin downregulation and decreased its interaction with CD2-associated protein and nephrin, as determined by Western analysis and co-immunoprecipitation. |
| 9 | 16541019 | Sema 3A induced a 10-fold increase in podocyte apoptosis and significantly decreased the activity of the Akt survival pathway. |
| 10 | 30236763 | Sema 3A as a biomarker of the activated mTOR pathway during hexavalent chromium-induced acute kidney injury. |
| 11 | 30236763 | Further, Cr(VI) treatment, in combination with pretreatment of the mTOR pathway inhibitor, Rap, showed a considerably stronger protective effect of Rap in protecting against Cr(VI)-induced nephrotoxicity than that seen with the free radical scavenger NAC, highlighting the dominant renal protective role of the mTOR pathway in inhibiting toxicity by downregulating the expressed levels of sema 3A in renal tissue. |
| 12 | 33828546 | It remains to be established if all these relations observed in patients with anti-PLA2R antibodies are also valid for expanding spectrum of antibodies directed against recently discovered minor antigens (e.g., THSD7A, NELL-1, semaphorin 3B). |