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Gene Information

Gene symbol: TRPV1

Gene name: transient receptor potential cation channel, subfamily V, member 1

HGNC ID: 12716

Related Genes

#	Gene Symbol	Number of hits
1	CLU	1 hits
2	FUNDC1	1 hits
3	PRKAA1	1 hits
4	TRPC6	1 hits
5	TRPM2	1 hits
6	TRPV4	1 hits

Related Sentences

#	PMID	Sentence
1	28028935	Renoprotection: focus on TRPV1, TRPV4, TRPC6 and TRPM2.
2	28028935	TRP melastatin (TRPM2) non-selective cation channels are expressed in the cytoplasm and intracellular organelles, where their inhibition ameliorates ischaemic renal pathology.
3	28028935	Although some of their basic properties have been recently identified, the renovascular role of TRPV1, TRPV4, TRPC6 and TRPM2 channels in disease states such as obesity, hypertension and diabetes is largely unknown.
4	28028935	In this review, we discuss recent evidence for TRPV1, TRPV4, TRPC6 and TRPM2 serving as potential targets for acute and chronic renoprotection in chronic vascular and metabolic disease.
5	28028935	Renoprotection: focus on TRPV1, TRPV4, TRPC6 and TRPM2.
6	28028935	TRP melastatin (TRPM2) non-selective cation channels are expressed in the cytoplasm and intracellular organelles, where their inhibition ameliorates ischaemic renal pathology.
7	28028935	Although some of their basic properties have been recently identified, the renovascular role of TRPV1, TRPV4, TRPC6 and TRPM2 channels in disease states such as obesity, hypertension and diabetes is largely unknown.
8	28028935	In this review, we discuss recent evidence for TRPV1, TRPV4, TRPC6 and TRPM2 serving as potential targets for acute and chronic renoprotection in chronic vascular and metabolic disease.
9	28028935	Renoprotection: focus on TRPV1, TRPV4, TRPC6 and TRPM2.
10	28028935	TRP melastatin (TRPM2) non-selective cation channels are expressed in the cytoplasm and intracellular organelles, where their inhibition ameliorates ischaemic renal pathology.
11	28028935	Although some of their basic properties have been recently identified, the renovascular role of TRPV1, TRPV4, TRPC6 and TRPM2 channels in disease states such as obesity, hypertension and diabetes is largely unknown.
12	28028935	In this review, we discuss recent evidence for TRPV1, TRPV4, TRPC6 and TRPM2 serving as potential targets for acute and chronic renoprotection in chronic vascular and metabolic disease.
13	32061660	Mechanistically, TRPV1-mediated transient Ca2+ influx activated 5' AMP-activated protein kinase (AMPK) that reduced the transcription of Fundc1, a key molecule participating in MAMs formation.
14	32061660	Inhibition of AMPK or overexpression of Fundc1 obviously blocked the inhibitory effect of capsaicin on MAMs formation and functional decline in podocytes.