Ignet

Gene Information

Gene symbol: ZNF580

Gene name: zinc finger protein 580

HGNC ID: 29473

Related Genes

#	Gene Symbol	Number of hits
1	NPHS1	1 hits

Related Sentences

#	PMID	Sentence
1	28606850	The impact of α-Lipoic acid on cell viability and expression of nephrin and ZNF580 in normal human podocytes.
2	28606850	Therefore, we analyzed the impact of α-LA on cell viability and expression of nephrin and zinc finger protein 580 (ZNF580) in normal hPC in vitro.
3	28606850	This was associated with reduced nephrin and ZNF580 expression and increased nephrin phosphorylation in normal hPC.
4	28606850	Moreover, α-LA reduced nephrin and ZNF580 protein expression via 'kappa-light-chain-enhancer' of activated B-cells (NF-κB) inhibition.
5	28606850	These data demonstrate that low α-LA had no negative influence on hPC viability, whereas, high α-LA concentrations induced cytotoxic effects on normal hPC and reduced nephrin and ZNF580 expression via NF-κB inhibition.
6	28606850	The impact of α-Lipoic acid on cell viability and expression of nephrin and ZNF580 in normal human podocytes.
7	28606850	Therefore, we analyzed the impact of α-LA on cell viability and expression of nephrin and zinc finger protein 580 (ZNF580) in normal hPC in vitro.
8	28606850	This was associated with reduced nephrin and ZNF580 expression and increased nephrin phosphorylation in normal hPC.
9	28606850	Moreover, α-LA reduced nephrin and ZNF580 protein expression via 'kappa-light-chain-enhancer' of activated B-cells (NF-κB) inhibition.
10	28606850	These data demonstrate that low α-LA had no negative influence on hPC viability, whereas, high α-LA concentrations induced cytotoxic effects on normal hPC and reduced nephrin and ZNF580 expression via NF-κB inhibition.
11	28606850	The impact of α-Lipoic acid on cell viability and expression of nephrin and ZNF580 in normal human podocytes.
12	28606850	Therefore, we analyzed the impact of α-LA on cell viability and expression of nephrin and zinc finger protein 580 (ZNF580) in normal hPC in vitro.
13	28606850	This was associated with reduced nephrin and ZNF580 expression and increased nephrin phosphorylation in normal hPC.
14	28606850	Moreover, α-LA reduced nephrin and ZNF580 protein expression via 'kappa-light-chain-enhancer' of activated B-cells (NF-κB) inhibition.
15	28606850	These data demonstrate that low α-LA had no negative influence on hPC viability, whereas, high α-LA concentrations induced cytotoxic effects on normal hPC and reduced nephrin and ZNF580 expression via NF-κB inhibition.
16	28606850	The impact of α-Lipoic acid on cell viability and expression of nephrin and ZNF580 in normal human podocytes.
17	28606850	Therefore, we analyzed the impact of α-LA on cell viability and expression of nephrin and zinc finger protein 580 (ZNF580) in normal hPC in vitro.
18	28606850	This was associated with reduced nephrin and ZNF580 expression and increased nephrin phosphorylation in normal hPC.
19	28606850	Moreover, α-LA reduced nephrin and ZNF580 protein expression via 'kappa-light-chain-enhancer' of activated B-cells (NF-κB) inhibition.
20	28606850	These data demonstrate that low α-LA had no negative influence on hPC viability, whereas, high α-LA concentrations induced cytotoxic effects on normal hPC and reduced nephrin and ZNF580 expression via NF-κB inhibition.
21	28606850	The impact of α-Lipoic acid on cell viability and expression of nephrin and ZNF580 in normal human podocytes.
22	28606850	Therefore, we analyzed the impact of α-LA on cell viability and expression of nephrin and zinc finger protein 580 (ZNF580) in normal hPC in vitro.
23	28606850	This was associated with reduced nephrin and ZNF580 expression and increased nephrin phosphorylation in normal hPC.
24	28606850	Moreover, α-LA reduced nephrin and ZNF580 protein expression via 'kappa-light-chain-enhancer' of activated B-cells (NF-κB) inhibition.
25	28606850	These data demonstrate that low α-LA had no negative influence on hPC viability, whereas, high α-LA concentrations induced cytotoxic effects on normal hPC and reduced nephrin and ZNF580 expression via NF-κB inhibition.