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Gene Information
Gene symbol: MYH7
Gene name: myosin, heavy chain 7, cardiac muscle, beta
HGNC ID: 7577
Synonyms: CMD1S
Related Genes
| # | Gene Symbol | Number of hits |
| 1 | DHFR | 1 hits |
| 2 | HLA-A | 1 hits |
| 3 | INS | 1 hits |
| 4 | MAPK1 | 1 hits |
| 5 | MSTN | 1 hits |
| 6 | MYH14 | 1 hits |
| 7 | SOD1 | 1 hits |
Related Sentences
| # | PMID | Sentence |
| 1 | 6232163 | Changes in the expression of different myosin heavy chain genes are most likely responsible for the thyroid hormone and insulin-induced alterations in myosin isoenzyme predominance. |
| 2 | 9176186 | Previous studies show that diabetes induces marked transformations in cardiac myosin heavy chain (MHC) gene expression that are somehow linked to the cellular action of thyroid hormone 3,5,3'-triiodothyronine (T3). |
| 3 | 11915909 | Using stable isotope tracer methodologies and mass spectrometric detection, we observed: (a) 76-92-year-old physically frail and 62-74-year-old middle-age adults have lower mixed muscle protein synthetic rates than 20-32-year-old men and women; (b) 2 weeks and 3 months of weightlifting exercise increased the synthetic rate of myosin heavy chain (MHC) and mixed muscle proteins to a similar magnitude in frail, middle-age, and young women and men; (c) Serum myostatin-immunoreactive protein levels were elevated in physically frail women and were inversely correlated with lean mass. |
| 4 | 18199585 | Diabetes depressed expression of Kv1.2 and dihydrofolate reductase (DHFR), increased beta-myosin heavy-chain expression, stimulated p38 MAPK, and reduced levels of total Akt and phosphorylated Akt/eNOS, all of which with the exception of myosin heavy chain were attenuated by IGF-I. |
| 5 | 20836552 | Additionally, a shift in cardiac myosin heavy chain (MHC) gene expression from ?- to ?-MHC isoform, decreased levels of superoxide dismutase-1 (SOD-1) and cardiac ?-actin, and elevated cardiac thiobarbituric acid reactive substances (TBARS) and caspase- and p38-NF?B-leading apoptosis signaling activities were demonstrated in diabetic hearts. |
| 6 | 16123338 | Mice with heart-specific overexpression of peroxisome proliferator-activated receptor (PPAR)alpha showed a metabolic and cardiomyopathic phenotype similar to the diabetic heart, and we determined tissue-specific glucose metabolism and insulin action in vivo during hyperinsulinemic-euglycemic clamps in awake myosin heavy chain (MHC)-PPARalpha mice (12-14 weeks of age). |